Title: TEACHING Homoeopathic approach in Thrombosis
1TEACHING Homoeopathic approach in Thrombosis
infarction
- DR R.Y NADAF
-
M.D (Hom) - PRINCIPAL
- Dr. B.D Jatti Homoeopathic Medical College
Hospital, Dharwad
2- To teach successfully,
- One must plan successfully.
- And successful planning means
- Knowing how to facilitate a positive
learning experience for all students.
3TEACHING
- The teacher should use his / her best
professional judgement to decide which method, - Strategy techniques which will work best for
particular situation.
4TEACHING
- . In recent years there has been increased
concern among practitioners and educational
researchers about practical side and
effectiveness of teaching
5TEACHING
- The how of teaching is now being given as
much significance as what and why in
academic circles
6TEACHING Pathology
- Pathology forms a core component of any
medical or dental undergraduate's course, yet it
is a subject that students often struggle with
due to the overwhelming amount of information
there is to assimilate in the time available.
7TEACHING Pathology
- Fundamental pathologic theory and principles
in co relation with General pathology of
Homoeopathy need to be explained in an
easy-to-read and engaging manner, using extensive
learning features to make learning as quick and
effective as possible
8TEACHING Pathology
- Dramatic curricular reforms in undergraduate
homoeopathic medical education mean that many
teachers now find themselves involved in courses
that are significantly different from those which
they encountered as medical students. - Department-led didactic courses in pathology have
been replaced by centrally managed, problem-based
integrated curricula in which pathology may at
first be difficult to identify.
9TEACHING Pathology
- Finally, consideration needs to be given
to the necessity for those involved in
homoeopathic medical education to be proficient
both in homoeopathic perspective and in
pathological basics.
10TEACHING Pathology
- The following are model slides an attempt
to teach thrombosis Infarction to students as
a pathological phenomenon its homoeopathic
perspective.
11Thrombosis Infarction
- A coronary thrombosis is seen
microscopically occluding the remaining small
lumen of this coronary artery. Such an acute
coronary thrombosis is often the antecedent to
acute myocardial infarction.
12What is Thrombosis ?
- Thrombosis (Greek ???µß?s??) is the
formation of a blood clot (thrombus) inside a
blood vessel, obstructing the flow of blood
through the circulatory system. When a blood
vessel is injured, the body uses platelets and
fibrin to form a blood clot, because the first
step in repairing it (hemostasis) is to prevent
loss of blood. If that mechanism causes too much
clotting, and the clot breaks free, an embolus is
formed
13Causes of Thrombosis
- In classical terms, thrombosis is
caused by abnormalities in one or more of the
following (Virchow's triad) The triad consists
of - 1. Alterations in normal blood flow2.
Injuries to the vascular endothelium3.
Alterations in the constitution of blood - (hypercoagulability)
14Virchows Triad
- 1) Alteration in blood flow can include
turbulence, - stasis, mitral stenosis, and varicose veins.
- 2)Injuries to the vascular endothelium can be
- cause by damage to the veins arising from
shear stress or hypertension. - 3)Hypercoagubility can be a consequence of
numerous possible risk factors such as
hyperviscosity, deficiency of antithrombin III,
nephrotic syndrome, changes after severe trauma
or burn, disseminated cancer, late pregnancy and
delivery, race, age, smoking, and obesity.
15Virchows Triad
16Classification
- There are two distinct forms of
thrombosis, each of which can be presented by
several subtypes - 1.Venous Thrombosis - is the formation of a
thrombus (blood clot) within a vein. - 2. Arterial thrombosis - is the formation of a
thrombus within an artery. In most cases,
arterial thrombosis follows rupture of atheroma,
and is therefore referred to as atherothrombosis
17Classification
- Venous thrombosis
- 1. Deep vein thrombosis
- 2. Portal vein thrombosis
- 3. Jugular vein thrombosis
- 4. Renal vein thrombosis
- 5. Budd Chiari Syndrome
- 6. Pagett Schrotter disease
- 7. Cerebral venous sinus thrombosis
18Classification
- 2 )Arterial Thrombosis
- 1. Stroke
- 2. Myocardial
- infarction
19Venous thrombosis
- 1.Deep vein thrombosis (DVT) is the
formation of a blood clot within a deep
vein. - - It most commonly affects leg veins, such
as the - femoral vein.
- - Three factors are important in the
formation of a blood clot within a deep
veinthese are the rate of blood flow, the
thickness of the blood and qualities of the
vessel wall. - - Classical signs of DVT include swelling,
pain and redness of the affected area.
20Venous thrombosis
- 2.Portal vein thrombosis - Portal vein thrombosis
is a form of venous thrombosis affecting the
hepatic portal vein, which can lead to portal
hypertension and reduction of the blood supply to
the liver. It usually has a pathological cause
such as pancreatitis, cirrhosis, diverticulitis
or cholangiocarcinoma
21Venous thrombosis
- 3.Jugular Vein Thrombosis - is a condition that
may occur due to infection, intravenous drug use
or malignancy. Jugular Vein Thrombosis can have a
varying list of complications, including
systemic sepsis, pulmonary embolism, and
papilledema. Characterized by a sharp pain at the
site of the vein, it's difficult to diagnose,
because it can occur at random.
22Venous thrombosis
- 4. Renal vein thrombosis - is the obstruction of
the renal vein by a thrombus. This tends to lead
to reduced drainage from the kidney.
23Venous thrombosis
- 5.Budd-Chiari syndrome is the blockage of the
hepatic vein or the inferior vena cava. This form
of thrombosis presents with abdominal pain,
ascites and hepatomegaly. Treatment varies
between drug therapy and surgical intervention by
the use of shunts.
24Venous thrombosis
- 6. Paget-Schroetter disease - is the obstruction
of an upper extremity vein (such as the axillary
vein or subclavian vein) by a thrombus. The
condition usually comes to light after vigorous
exercise and usually presents in younger,
otherwise healthy people. Men are affected more
than women
25Venous thrombosis
- 7. Cerebral venous sinus thrombosis (CVST) - is a
rare form of stroke which results from the
blockage of the dural venous sinuses by a
thrombus. Symptoms may include headache, abnormal
vision, any of the symptoms of stroke such as
weakness of the face and limbs on one side of the
body and seizures. The diagnosis is usually made
with a CT or MRI scan. The majority of persons
affected make a full recovery.
26Arterial thrombosis - Stroke
- 1.Stroke - is the rapid decline of brain
function due to a disturbance in the supply of
blood to the brain. This can be due to ischemia,
thrombus, embolus (a lodged particle) or
hemorrhage (a bleed). In thrombotic stroke, a
thrombus (blood clot) usually forms around
atherosclerotic plaques. Since blockage of the
artery is gradual, onset of symptomatic
thrombotic strokes is slower.
272. Arterial thrombosis - Stroke
- Thrombotic stroke can be divided into two
categorieslarge vessel disease and small vessel
disease. The former affects vessels such as the
internal carotids, vertebral and the circle of
Willis. The latter can affect smaller vessels
such as the branches of the circle of Willis.
282. Arterial thrombosis
- Myocardial infarction (MI) is caused by an
infarct (death of tissue due to ischemia), often
due to the obstruction of the coronary artery by
a thrombus. MI can quickly become fatal if
emergency medical treatment is not received
promptly .
29What is infarction ?
- An infarction is the process of tissue
death (necrosis) caused by blockage of the
tissue's blood supply. - The supplying artery may be blocked by an
obstruction (e.g. an embolus, thrombus, or
atherosclerotic plaque), - may be mechanically compressed (e.g. tumor,
volvulus, or hernia), ruptured by trauma (e.g.
atherosclerosis or vasculitides), or - vasoconstricted (e.g. cocaine vasoconstriction
leading to myocardial infarction).
30Infarctions
- Infarctions are commonly associated with
hypertension or atherosclerosis. In
atherosclerotic formations a plaque develops
under a fibrous cap. When the fibrous cap is
degraded by metalloproteinases released from
macrophages or by intravascular shear force from
blood flow subendothelial thrombogenic material
(extracellular matrix) is exposed to circulating
platelets and thrombus formation occurs on the
vessel wall occluding blood flow.
31Infarctions
- Occasionally, the plaque may rupture
forming an embolus that travels with the blood
flow downstream where the vessel narrows and
eventually clogs the vessel lumen. Infarctions
can also involve mechanical blockage of the blood
supply, such as when part of the gut or testicles
herniates or becomes involved in a volvulus.
32Classification of infarction
- Infarctions are divided into 2 types
according to the amount of blood present - 1.White infarctions (anemic infarcts)
- 2.Red infarctions (hemorrhagic infarcts)
-
331.White infarctions (anemic infarcts)
- White infarctions (anemic infarcts)
affect solid organs such as the heart, spleen and
kidneys wherein the solidity of the tissue
(biology) substantially limits the amount of
nutrients (blood/oxygen/glucose/fuel) that can
flow into the area of ischemic necrosis. Similar
occlusion to blood flow and consequent necrosis
can occur as a result of severe vasoconstriction
as illustrated in severe Raynaud's phenomenon
that can lead to irreversible gangrene.
342. Red infarctions (hemorrhagic infarcts)
- Red infarctions (hemorrhagic infarcts),
generally affect the lungs or other loose organs
(testis, ovary, small intestines). The occlusion
consists more of red blood cells and fibrin
strands. Characteristics of red infarcts include
occlusion of a vein loose tissues that allow
blood to collect in the infarcted zone tissues
with a dual circulatory system (lung, small
intestines) tissues previously congested from
sluggish venous outflow and reperfusion (injury)
of previously ischemic tissue that is associated
with reperfusion-related diseases such as -
Myocardial infarction, stroke (cerebral
infarction), shock-resuscitation, replantation
surgery, frostbite, burns and organ
transplantation
35Diseases commonly associated with infarctions
include
- 1. Myocardial infarction (heart attack)
- 2. Pulmonary embolism ("lung attack")
- 3.Cerebral infarction (stroke)
- 4.Peripheral artery occlusive disease (the
most - severe form of which is gangrene)
- 5.Antiphospholipid syndrome
- 6.Sepsis
- 7.Giant-cell arteritis (GCA)
- 8.Hernia
- 9.Volvulus
- 10.Splenic infarction
36Homoeopathic Perspective
- Therapeutic nihilism( travesty of medicine)
originated with that group of pathologists (not
practicing physicians) who sought to identify
every disorder disease with definite anatomical
changes. They led clinicians to study disease
only in this relation. The fact is that
anatomical changes are results of disease not
the disease process itself.
37Homoeopathic Perspective
- Disturbed physiology always precedes
pathology but does not always produce it.
Therefore, symptoms present themselves, before
during , as well as after the formation of
pathological end products or tissue changes. The
homoeopathic prescriber utilizes all signs
symptoms but recognizes their relative importance
38Hahnemannian concept of disease phenomenon
- Disease is primarily a disturbance in the
vital force which governs regulates all the
organs parts of the body. In health this vital
force maintains normal growth co-ordination of
all organic functions. when, from some
disease-producing cause, this force becomes
disturbed, disease or disharmony of function
results. The cause of disturbance may be
infections, injuries, climatic conditions,
violent emotions, dietary errors etc.
39Understanding pathology in homoeopathic parlance
- A true science of pathology must include be
able to explain all the symptoms of disease -
the finer, subjective individual symptoms as well
as the general functional, organic objective
changes that occur in a disease. - A true science of therapeutics must be able to
explain all the symptoms of disease the finer,
40MIASM- Fundamental cause Each and every
individual receives a number of inputs through
out his/her life every one reacts differently
to each of these inputs.
H
Input
Output
41- The reactions
- are mainly
- dependant on
- Heredity
- Genetic pattern and
- The environmental influences that one
- experiences in every stage of ones life.
42Miasmatic cleavage of thrombosis Infarction
- From the structural point of view, thrombosis is
an expression of an actual change of structure in
the arterial system. - Change of structure occurs in complicated Psora,
i.e when the psoric dyscrasia is combined with
syphillitic, structural changes do take place,
in this manifestation we have an expression of
the combination of the two stigmata.
43Miasmatic cleavage of thrombosis Infarction
- Syphillitic dyscrasia befits the expression of
infarction. Infarction is a process of tissue
death infarctions are commonly associated with
hypertension or atherosclerosis which contribute
to gross structural changes. - White infarcts are more of functional distortion
culminating into death of tissues due to lack of
blood supply.Hence psora to syphillis. - Red infarcts occur in previously ischemic tissue
that is associated with reperfusion-related
diseases such as - Myocardial infarction, stroke
(cerebral infarction) the tissues are under the
deadning influence of syphillitic dyscrasia.
44Treatment Protocol
- Totalistic ,Holistic, or Constitutional treatment
in tandem with life style changes is the basis
of prescription in the homoeopathic management of
Thrombosis Infarction. - Role of homoeopathy is more in Preventive
Cardiology Post infarction Management.
Homoeopathic medicines can control prevent
causes of IHD
45Therapeutics - Thrombosis
- Mainly indicated remedies are Apis, Ars, Both-l,
Lach, Kali-m, Nat-s, and Vip. Aconitum napellus
Arnica montana Belladonna atropa Bothrops
lanceolatus Hamamelis virginica Lachesis
Pulsatilla nigricans.
46 Therapeutics -Cerebral Haemorrhage, Embolism
and Thrombosis -
- Opium, Belladonna, Arnica, Laurocerasus are the
remedies undoubtedly most similar to the general
symptoms of apoplexy
47Therapeutics Myocardial infarction
- Aconite, Cactus grandiflora,Glonine,
- Camphor, Digitalis, Arnica, Rhus tox
48Thank U!