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Title: TEACHING Homoeopathic approach in Thrombosis


1
TEACHING Homoeopathic approach in Thrombosis
infarction
  • DR R.Y NADAF

  • M.D (Hom)
  • PRINCIPAL
  • Dr. B.D Jatti Homoeopathic Medical College
    Hospital, Dharwad

2
  • To teach successfully,
  • One must plan successfully.
  • And successful planning means
  • Knowing how to facilitate a positive
    learning experience for all students.

3
TEACHING
  • The teacher should use his / her best
    professional judgement to decide which method,
  • Strategy techniques which will work best for
    particular situation.

4
TEACHING
  • . In recent years there has been increased
    concern among practitioners and educational
    researchers about practical side and
    effectiveness of teaching

5
TEACHING
  • The how of teaching is now being given as
    much significance as what and why in
    academic circles

6
TEACHING Pathology
  • Pathology forms a core component of any
    medical or dental undergraduate's course, yet it
    is a subject that students often struggle with
    due to the overwhelming amount of information
    there is to assimilate in the time available.

7
TEACHING Pathology
  • Fundamental pathologic theory and principles
    in co relation with General pathology of
    Homoeopathy need to be explained in an
    easy-to-read and engaging manner, using extensive
    learning features to make learning as quick and
    effective as possible

8
TEACHING Pathology
  • Dramatic curricular reforms in undergraduate
    homoeopathic medical education mean that many
    teachers now find themselves involved in courses
    that are significantly different from those which
    they encountered as medical students.
  • Department-led didactic courses in pathology have
    been replaced by centrally managed, problem-based
    integrated curricula in which pathology may at
    first be difficult to identify.

9
TEACHING Pathology
  • Finally, consideration needs to be given
    to the necessity for those involved in
    homoeopathic medical education to be proficient
    both in homoeopathic perspective and in
    pathological basics.

10
TEACHING Pathology
  • The following are model slides an attempt
    to teach thrombosis Infarction to students as
    a pathological phenomenon its homoeopathic
    perspective.

11
Thrombosis Infarction
  • A coronary thrombosis is seen
    microscopically occluding the remaining small
    lumen of this coronary artery. Such an acute
    coronary thrombosis is often the antecedent to
    acute myocardial infarction.

12
What is Thrombosis ?
  • Thrombosis (Greek ???µß?s??) is the
    formation of a blood clot (thrombus) inside a
    blood vessel, obstructing the flow of blood
    through the circulatory system. When a blood
    vessel is injured, the body uses platelets and
    fibrin to form a blood clot, because the first
    step in repairing it (hemostasis) is to prevent
    loss of blood. If that mechanism causes too much
    clotting, and the clot breaks free, an embolus is
    formed

13
Causes of Thrombosis
  • In classical terms, thrombosis is
    caused by abnormalities in one or more of the
    following (Virchow's triad) The triad consists
    of
  • 1. Alterations in normal blood flow2.
    Injuries to the vascular endothelium3.
    Alterations in the constitution of blood
  • (hypercoagulability)

14
Virchows Triad
  • 1) Alteration in blood flow can include
    turbulence,
  • stasis, mitral stenosis, and varicose veins.
  • 2)Injuries to the vascular endothelium can be
  • cause by damage to the veins arising from
    shear stress or hypertension.
  • 3)Hypercoagubility can be a consequence of
    numerous possible risk factors such as
    hyperviscosity, deficiency of antithrombin III,
    nephrotic syndrome, changes after severe trauma
    or burn, disseminated cancer, late pregnancy and
    delivery, race, age, smoking, and obesity.

15
Virchows Triad
16
Classification
  • There are two distinct forms of
    thrombosis, each of which can be presented by
    several subtypes
  • 1.Venous Thrombosis - is the formation of a
    thrombus (blood clot) within a vein.
  • 2. Arterial thrombosis - is the formation of a
    thrombus within an artery. In most cases,
    arterial thrombosis follows rupture of atheroma,
    and is therefore referred to as atherothrombosis

17
Classification
  • Venous thrombosis
  • 1. Deep vein thrombosis
  • 2. Portal vein thrombosis
  • 3. Jugular vein thrombosis
  • 4. Renal vein thrombosis
  • 5. Budd Chiari Syndrome
  • 6. Pagett Schrotter disease
  • 7. Cerebral venous sinus thrombosis

18
Classification
  • 2 )Arterial Thrombosis
  • 1. Stroke
  • 2. Myocardial
  • infarction

19
Venous thrombosis
  • 1.Deep vein thrombosis (DVT) is the
    formation of a blood clot within a deep
    vein.
  • - It most commonly affects leg veins, such
    as the
  • femoral vein.
  • - Three factors are important in the
    formation of a blood clot within a deep
    veinthese are the rate of blood flow, the
    thickness of the blood and qualities of the
    vessel wall.
  • - Classical signs of DVT include swelling,
    pain and redness of the affected area.

20
Venous thrombosis
  • 2.Portal vein thrombosis - Portal vein thrombosis
    is a form of venous thrombosis affecting the
    hepatic portal vein, which can lead to portal
    hypertension and reduction of the blood supply to
    the liver. It usually has a pathological cause
    such as pancreatitis, cirrhosis, diverticulitis
    or cholangiocarcinoma

21
Venous thrombosis
  • 3.Jugular Vein Thrombosis - is a condition that
    may occur due to infection, intravenous drug use
    or malignancy. Jugular Vein Thrombosis can have a
    varying list of complications, including
    systemic sepsis, pulmonary embolism, and
    papilledema. Characterized by a sharp pain at the
    site of the vein, it's difficult to diagnose,
    because it can occur at random.

22
Venous thrombosis
  • 4. Renal vein thrombosis - is the obstruction of
    the renal vein by a thrombus. This tends to lead
    to reduced drainage from the kidney.

23
Venous thrombosis
  • 5.Budd-Chiari syndrome is the blockage of the
    hepatic vein or the inferior vena cava. This form
    of thrombosis presents with abdominal pain,
    ascites and hepatomegaly. Treatment varies
    between drug therapy and surgical intervention by
    the use of shunts.

24
Venous thrombosis
  • 6. Paget-Schroetter disease - is the obstruction
    of an upper extremity vein (such as the axillary
    vein or subclavian vein) by a thrombus. The
    condition usually comes to light after vigorous
    exercise and usually presents in younger,
    otherwise healthy people. Men are affected more
    than women

25
Venous thrombosis
  • 7. Cerebral venous sinus thrombosis (CVST) - is a
    rare form of stroke which results from the
    blockage of the dural venous sinuses by a
    thrombus. Symptoms may include headache, abnormal
    vision, any of the symptoms of stroke such as
    weakness of the face and limbs on one side of the
    body and seizures. The diagnosis is usually made
    with a CT or MRI scan. The majority of persons
    affected make a full recovery.

26
Arterial thrombosis - Stroke
  • 1.Stroke - is the rapid decline of brain
    function due to a disturbance in the supply of
    blood to the brain. This can be due to ischemia,
    thrombus, embolus (a lodged particle) or
    hemorrhage (a bleed). In thrombotic stroke, a
    thrombus (blood clot) usually forms around
    atherosclerotic plaques. Since blockage of the
    artery is gradual, onset of symptomatic
    thrombotic strokes is slower.

27
2. Arterial thrombosis - Stroke
  • Thrombotic stroke can be divided into two
    categorieslarge vessel disease and small vessel
    disease. The former affects vessels such as the
    internal carotids, vertebral and the circle of
    Willis. The latter can affect smaller vessels
    such as the branches of the circle of Willis.

28
2. Arterial thrombosis
  • Myocardial infarction (MI) is caused by an
    infarct (death of tissue due to ischemia), often
    due to the obstruction of the coronary artery by
    a thrombus. MI can quickly become fatal if
    emergency medical treatment is not received
    promptly .

29
What is infarction ?
  • An infarction is the process of tissue
    death (necrosis) caused by blockage of the
    tissue's blood supply.
  • The supplying artery may be blocked by an
    obstruction (e.g. an embolus, thrombus, or
    atherosclerotic plaque),
  • may be mechanically compressed (e.g. tumor,
    volvulus, or hernia), ruptured by trauma (e.g.
    atherosclerosis or vasculitides), or
  • vasoconstricted (e.g. cocaine vasoconstriction
    leading to myocardial infarction).

30
Infarctions
  • Infarctions are commonly associated with
    hypertension or atherosclerosis. In
    atherosclerotic formations a plaque develops
    under a fibrous cap. When the fibrous cap is
    degraded by metalloproteinases released from
    macrophages or by intravascular shear force from
    blood flow subendothelial thrombogenic material
    (extracellular matrix) is exposed to circulating
    platelets and thrombus formation occurs on the
    vessel wall occluding blood flow.

31
Infarctions
  • Occasionally, the plaque may rupture
    forming an embolus that travels with the blood
    flow downstream where the vessel narrows and
    eventually clogs the vessel lumen. Infarctions
    can also involve mechanical blockage of the blood
    supply, such as when part of the gut or testicles
    herniates or becomes involved in a volvulus.

32
Classification of infarction
  • Infarctions are divided into 2 types
    according to the amount of blood present
  • 1.White infarctions (anemic infarcts)
  • 2.Red infarctions (hemorrhagic infarcts)

33
1.White infarctions (anemic infarcts)
  • White infarctions (anemic infarcts)
    affect solid organs such as the heart, spleen and
    kidneys wherein the solidity of the tissue
    (biology) substantially limits the amount of
    nutrients (blood/oxygen/glucose/fuel) that can
    flow into the area of ischemic necrosis. Similar
    occlusion to blood flow and consequent necrosis
    can occur as a result of severe vasoconstriction
    as illustrated in severe Raynaud's phenomenon
    that can lead to irreversible gangrene.

34
2. Red infarctions (hemorrhagic infarcts)
  • Red infarctions (hemorrhagic infarcts),
    generally affect the lungs or other loose organs
    (testis, ovary, small intestines). The occlusion
    consists more of red blood cells and fibrin
    strands. Characteristics of red infarcts include
    occlusion of a vein loose tissues that allow
    blood to collect in the infarcted zone tissues
    with a dual circulatory system (lung, small
    intestines) tissues previously congested from
    sluggish venous outflow and reperfusion (injury)
    of previously ischemic tissue that is associated
    with reperfusion-related diseases such as -
    Myocardial infarction, stroke (cerebral
    infarction), shock-resuscitation, replantation
    surgery, frostbite, burns and organ
    transplantation

35
Diseases commonly associated with infarctions
include
  • 1. Myocardial infarction (heart attack)
  • 2. Pulmonary embolism ("lung attack")
  • 3.Cerebral infarction (stroke)
  • 4.Peripheral artery occlusive disease (the
    most
  • severe form of which is gangrene)
  • 5.Antiphospholipid syndrome
  • 6.Sepsis
  • 7.Giant-cell arteritis (GCA)
  • 8.Hernia
  • 9.Volvulus
  • 10.Splenic infarction

36
Homoeopathic Perspective
  • Therapeutic nihilism( travesty of medicine)
    originated with that group of pathologists (not
    practicing physicians) who sought to identify
    every disorder disease with definite anatomical
    changes. They led clinicians to study disease
    only in this relation. The fact is that
    anatomical changes are results of disease not
    the disease process itself.

37
Homoeopathic Perspective
  • Disturbed physiology always precedes
    pathology but does not always produce it.
    Therefore, symptoms present themselves, before
    during , as well as after the formation of
    pathological end products or tissue changes. The
    homoeopathic prescriber utilizes all signs
    symptoms but recognizes their relative importance

38
Hahnemannian concept of disease phenomenon
  • Disease is primarily a disturbance in the
    vital force which governs regulates all the
    organs parts of the body. In health this vital
    force maintains normal growth co-ordination of
    all organic functions. when, from some
    disease-producing cause, this force becomes
    disturbed, disease or disharmony of function
    results. The cause of disturbance may be
    infections, injuries, climatic conditions,
    violent emotions, dietary errors etc.

39
Understanding pathology in homoeopathic parlance
  • A true science of pathology must include be
    able to explain all the symptoms of disease -
    the finer, subjective individual symptoms as well
    as the general functional, organic objective
    changes that occur in a disease.
  • A true science of therapeutics must be able to
    explain all the symptoms of disease the finer,

40
MIASM- Fundamental cause Each and every
individual receives a number of inputs through
out his/her life every one reacts differently
to each of these inputs.
H
Input
Output
41
  • The reactions
  • are mainly
  • dependant on
  • Heredity
  • Genetic pattern and
  • The environmental influences that one
  • experiences in every stage of ones life.

42
Miasmatic cleavage of thrombosis Infarction
  • From the structural point of view, thrombosis is
    an expression of an actual change of structure in
    the arterial system.
  • Change of structure occurs in complicated Psora,
    i.e when the psoric dyscrasia is combined with
    syphillitic, structural changes do take place,
    in this manifestation we have an expression of
    the combination of the two stigmata.

43
Miasmatic cleavage of thrombosis Infarction
  • Syphillitic dyscrasia befits the expression of
    infarction. Infarction is a process of tissue
    death infarctions are commonly associated with
    hypertension or atherosclerosis which contribute
    to gross structural changes.
  • White infarcts are more of functional distortion
    culminating into death of tissues due to lack of
    blood supply.Hence psora to syphillis.
  • Red infarcts occur in previously ischemic tissue
    that is associated with reperfusion-related
    diseases such as - Myocardial infarction, stroke
    (cerebral infarction) the tissues are under the
    deadning influence of syphillitic dyscrasia.

44
Treatment Protocol
  • Totalistic ,Holistic, or Constitutional treatment
    in tandem with life style changes is the basis
    of prescription in the homoeopathic management of
    Thrombosis Infarction.
  • Role of homoeopathy is more in Preventive
    Cardiology Post infarction Management.
    Homoeopathic medicines can control prevent
    causes of IHD

45
Therapeutics - Thrombosis
  • Mainly indicated remedies are Apis, Ars, Both-l,
    Lach, Kali-m, Nat-s, and Vip. Aconitum napellus
    Arnica montana Belladonna atropa Bothrops
    lanceolatus Hamamelis virginica Lachesis
    Pulsatilla nigricans.

46
Therapeutics -Cerebral Haemorrhage, Embolism
and Thrombosis -
  • Opium, Belladonna, Arnica, Laurocerasus are the
    remedies undoubtedly most similar to the general
    symptoms of apoplexy

47
Therapeutics Myocardial infarction
  • Aconite, Cactus grandiflora,Glonine,
  • Camphor, Digitalis, Arnica, Rhus tox

48
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