Title: Nasal Deformities
1Nasal Deformities
Septal Deviation
A congenital or traumatically acquired
bending or bowing of the nasal septum
2Symptoms
- Mild forms do not cause
- symptoms and have no pathologic significance
3Symptoms
- More pronounced degrees of septal curvature can
obstruct nasal breathing and may also cause
olfactory impairment due to inadequate
ventilation of the olfactory groove. -
- Deficient nasal airflow can also lead to
paranasal sinus sequelae such as headaches and
recurrent sinusitis. -
- A large septal spur that comes into
- contact with the nasal turbinates can
cause epistaxis
4Diagnosis
- Septal subluxation is a special form in which the
anterior septal margin is displaced from the
median plane. This condition is readily
identified by external inspection of the nasal
base. - Further clinical examination consists of anterior
rhinoscopy or endoscopy. - The degree of nasal obstruction can be
objectively evaluated by rhinomanometry. - For medicolegal reasons, olfactory testing should
always be done prior to surgical treatment
5Treatment
- The treatment of choice is surgical straightening
of the deviated septum (septoplasty)
6Deformities of the External Nose
Causes and forms
Deformities may be congenital or traumatically
acquired The most common deformities are a
crooked nose, humped nose, saddle nose, and broad
nose, which may occur separately or in
combinations
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8Diagnosis
- Inspection
- Anterior rhinoscopy
-
- Endoscopy
9Treatment
- The treatment of choice is functional
septorhinoplasty, with correction of the nasal
septum and external nose
10Nosebleed (Epistaxis)
- Nosebleed is a relatively common, usually
harmless symptom that may reflect a number of
diseases of variable severity
11Local causes
- Perforation
- traumatic
- iatrogenic
- Inflammatory
- spurs or ridges
- Foreign bodies
- rhinoliths
- trauma (including nose picking)
- allergy
- acute rhinitis
- Traumatic aneurysm of the internal carotid
- Benign neoplasms
- malignant neoplasms
12Systemic disease
- Atherosclerosis
- Infection
- Pregnancy
- Diabetes mellitus
- Congenital e.g., hemophilia A and B, Willebrand
disease - Acquired e.g., anticoagulant therapy,
- Hepatocellular insufficiency
- Platelet disorders
- Congenital
- Acquired uremia, dysproteinemia, adverse effects
of dextrann and acetylsalicylic acid (ASA)
therapy SchönleinHenoch purpura - Osler disease
13Diagnosis
- Nosebleed requires a simultaneous, coordinated
protocol of diagnostic and therapeutic actions
14Evaluation
- The diagnostic work-up begins with blood pressure
measurement. - Except in very minor cases, the Hb should also be
determined, and a coagulation disorder should be
excluded by determining the platelet count,
bleeding time, thromboplastin time, partial
thromboplastin time (PTT), and thrombin time
15Bleeding Site
- The nasal cavity is inspected by anterior
rhinoscopy or endoscopy following decongestion
and local anesthesia of the mucosa. - In most cases the bleeding site is in
Kiesselbachs area
16Treatment
- General measures
- The nostrils are compressed against the nasal
septum - the patient is told not to swallow blood running
down the pharynx. - The patient is kept in an upright posture
- An ice bag can be placed on the back of the neck
to induce reflex vasoconstriction - An intravenous line should be placed if bleeding
is severe
17Silver nitrate cautery
- Mild epistaxis from Kiesselbachs area can often
be controlled by selective local cauterization
18Nasal packing
- For severe epistaxis, the anterior nasal cavity
can be packed with ointment-impregnated gauze
strips
19Vascular ligation or embolization
- The most common source of bleeding from the
posterolateral part of the nasal cavity is the
sphenopalatine artery (branch of the maxillary
artery), which can be coagulated or clipped under
endoscopic control
20Surgical prevention of recurrent epistaxis
- The main indications for surgery are changes in
the nasal septum such as septal spurs, ridges,
and perforations. -
- Treatment consists of straightening the nasal
septum (septoplasty or closing the septal
perforation (e.g., by implanting an auricular
cartilage graft and using local mucosal flap
advancement
21Fractures of the Nasal Pyramid and Lateral Midface
22Nasal Pyramid Fracture
- The nasal pyramid is predisposed to fractures
because of its exposed location.
23Diagnostic procedure
24Palpation
- Crepitus noted on palpation confirms the
suspicion of a fracture
25Imaging
- Further diagnostic measures include radiographs
of the nose in the lateral projection - Standard sinus projections to exclude bony
involvement of the lateral midface
26Complications
- Subperichondrial hemorrhage with hematoma
- Septal Abcess
27Lateral Midfacial Fractures
- Lateral midfacial fractures are usually caused by
blunt trauma to the side of the face. -
- Affected structures of the bony facial skeleton
are the maxillary sinus, orbit, and the zygoma or
zygomatic arch
28- An isolated fracture of the orbital floor with a
partial herniation of the orbital contents into
the maxillary sinus is a special type of lateral
midfacial fracture called a blow-out fracture
29Symptoms
- Facial asymmetry
- Limited mouth opening
- Diplopia
- Sensory disturbances
30Diagnosis
- Inspection
- Swelling
- subcutaneous hemorrhage
- Asymmetry of the affected facial
- Enophthalmos
31- Palpation
- Concomitant soft-tissue swelling can make it
difficult or impossible to palpate sites of bony
discontinuity or displacement
32- Sensory testing
- Wisps of cotton can be used to test sensory
function on the healthy and affected sides
33- Radiographs
- Whenever a lateral midfacial fracture is
suspected, standard sinus radiographs should be
obtained (occipitomental and occipitofrontal
projections to define the extent of the bony
discontinuity or displacement
34- The zygomatic arches may be poorly visualized in
standard projections, and so a bucket handle
view should be added when a concomitant zygomatic
arch fracture is suspected
35- CT Scans
- be helpful to obtain a more discriminating view
of the fracture and also to exclude an
involvement of the anterior skull base
36Inspection
37Imaging
38Imaging
39Treatment
- Surgical treatment
- is unnecessary for undisplaced, asymptomatic
fractures - is indicated for displaced fractures or fractures
that are causing symptoms such as sensory
deficits in the distribution of the infraorbital
nerve, diplopia on upward gaze, enophthalmos,
restricted jaw opening, or facial asymmetry. - Treatment consists of reduction and fixation of
the bone fragments using miniplates, interosseous
wiring, or both
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41Fractures of the Central Midfaceand Anterior
Skull Base
42Classification
- Central midfacial fractures (Le Fort I-III)
- Frontobasal fractures (Escher classification)
43Le Fort classification
44Escher classification
45Notice
- Frontobasal fractures occupy a special place
among skull fractures because they are usually an
indirectly open injury that creates a
communication between the cranial cavity and the
environment lead to life-threatening intracranial
complications (e.g., meningitis, brain abscess)
46Symptoms
- Unilateral or bilateral periorbital hematoma
- Dish face the midface has been separated from
the skull base and displaced inward - Cerebrospinal fluid (CSF) rhinorrhea
- Vision loss
- Diplopia
- Cerebral prolapse
- Anosmia
47Examination
48CSF Leakage
49Detection of CSF
- Glucose test
- ß2-transferrin
50Diagnosis
- Computed tomography
- Axial scans are for evaluating the anterior and
posterior walls of the frontal sinuses and
sphenoid sinus - Coronal scans more clearly define the ethmoid
roof and cribriform plate
51Complementary tests
- Testing of hearing and balance
- Olfactory testing
52Treatment
- Every confirmed fracture of the anterior skull
base should be treated surgically in operable
patients, regardless of whether or not a CSF leak
has been detected
53Indications for surgery
54Vital indications (operate immediately)
- Life-threatening rise of intracranial pressure
due to intracranial hemorrhage - Bleeding from the nose or sinuses that is
refractory to conservative treatment - Bleeding from an open skull injury that is
refractory to conservative treatment
55Absolute indications (operate as soon as
possible)
- Open brain injury
- Dural tear from an indirectly open head injury
- Penetrating foreign bodies and impalement
injuries - Early complications (e.g., meningitis,
encephalitis, brain abscess) - Late complications (e.g., meningitis, brain
abscess, osteomyelitis) - Orbital complications
56Relative indications (operate in 12 weeks)
- Displaced bone fragments
- Fractures involving the drainage tracts of the
paranasal sinuses (ostiomeatal unit) - Acute or chronic sinusitis at the time of the
injury - Post-traumatic sinus inflammation, mucopyocele
formation - Supraorbital nerve injury due to an adjacent
fracture
57Questions
- Define the paradoxical cyanosis.
- Name four common nasal deformity.
- Where is the common site of epistaxis in old age?
- What is the most definitive sign for nasal
fracture? - Name six common symptoms for frontobasal
fracture.
58Session 4
- Inflammations of the External Nose, Nasal Cavity,
- and Facial Soft Tissues
59Inflammations of the Hair Follicles
- Folliculitis the disease is confined to the hair
follicles. - Furuncle the infection spreads to deeper
tissues and forms a central core of purulent
liquefaction.
60Symptoms
- Nasal furuncles present as painful, tender,
erythematous swellings about the nasal tip and
nares
61Treatment
- Antibiotic that is active against staphylococci
- Dicloxacillin sodium , Cephalexin and so on
- Combined with the local application of an
antibiotic-containing ointment
62Complications
- Inadequate treatment or manipulations of the
nasal furuncle itself can result in - Hematogenous spread to intracranial
structures
63Erysipelas
- Causative organisms are beta-hemolytic group A
streptococci - Less common pathogens are streptococci of other
groups, Staphylococcus aureus, and gram-negative
rods (e.g., Klebsiella pneumoniae)
64Symptoms
- High fever
- Feeling of tension in the soft tissues
- Rapidly by broad areas of erythema and swelling,
which are sharply demarcated from unaffected skin - The tissue is warm to the touch, and small
blisters occasionally form
65Treatment
- The treatment of choice is the parenteral
administration of penicillin
66Inflammations of the Nasal Cavity
67Acute Rhinitis
- Acute rhinitis (common cold) is the most
prevalent infectious disease - Rhinoviruses and coronaviruses comprise almost
half of the causative organisms of acute viral
rhinitis
68Symptoms
- Dry stage
- Malaise (lethargy, headache, fever) and local
discomfort in the nose and nasopharynx (burning,
soreness). - Catarrhal stage
- Watery, initially serous nasal discharge and
nasal obstruction due to mucosal swelling, which
mainly involves the turbinates.
69Bacterial Superinfection
- Viral damage to the epithelium promotes bacterial
colonization, which alters the consistency of the
clear nasal discharge, causing it to become
mucopurulent.
70Treatment
- Treatment consists of supportive measures to
relieve nasal obstruction and prevent sinusitis
and other sequelae by the use of decongestant
nose drops - Antibiotics may also be prescribed in patients
with bacterial superinfection or paranasal sinus
involvement
71Nonspecific Chronic Rhinitis
- Nonspecific chronic rhinitis can develop due to
anatomic changes (e.g., marked septal deviation,
septal spur) or other lesions of the nasal cavity
(polyps, tumors) and nasopharynx (adenoids) - Environmental factors such as sustained extreme
temperatures or air pollutants can also bring on
this condition
72Symptoms
- Patients present clinically with
- Obstructed nasal breathing
- Mucous nasal discharge
- Frequent throat clearing and occasional
hoarseness
73Treatment
- The most important step is to eliminate the cause
by removing chronic irritants from the
environment or by surgically correcting any
intranasal pathology (e.g., septoplasty)
74Specific Chronic Rhinitis
- Tuberculosis
- Sarcoidosis
- Rhinoscleroma
- Actinomycosis
- Syphilis
- Wegner Granulomatosis
- Fungal infections
- Aspergillosis
- Mucormycosis
- Rhinosporidiosis
75Allergic Rhinitis
- Triggered by an immediate, IgE-mediated reaction
of the immune system to any of a number of
foreign substances, particularly pollens and
animal allergens.
76Seasonal allergic rhinitis
- Mainly by pollens
- Disappear at the end of the pollen season
77Perennial allergic rhinitis
- Is caused by year-round allergen exposure
- The predominant causative allergens are house
dust, pet dander, and molds - The disease may also be caused by certain foods
(e.g., strawberries, nuts, eggs, fish) as well as
occupational exposure to allergens (e.g., bakers
and hairdressers)
78Symptoms
- The clinical manifestations
- Obstructed nasal breathing
- Sneezing attacks
- Watery nasal discharge
- Itching of the nose and eyes (conjunctivitis)
79Diagnosis
- Detailed allergy history (do the symptoms present
year-round or only during contact with certain
animals or plants). - Seasonal allergic rhinitis, a bluish-purple
discoloration of the mucosa. - Perennial rhinitis, the mucosa is bright red and
shows inflammatory changes. - Careful allergy testing is necessary to identify
the antigens involved.
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81Treatment
- The best treatment strategy is to avoid contact
with the allergen or eliminate allergenic
irritants - Pharmacologic treatment
- Mast-cell stabilizers
- Local and systemic H1 antihistamines
- Local steroids
- Immunotherapy or hyposensitization therapy
- Surgical options
82Vasomotor Rhinitis
- Resembles allergic rhinitis in its clinical
features, but there is no evidence that the
patient has been previously sensitized. - Neurovascular autonomic disturbances in
regulating the tonus of the nasal mucosal vessels
83Symptoms
- Obstructed nasal breathing
- Watery nasal discharge
- Sneezing
- The history shows that the symptoms are related
to a temperature change, the consumption of hot
liquid or alcohol, or less specifically to
emotional stress.
84Treatment
- Medical therapy includes
- Antihistamines
- corticosteroid-containing nasal sprays
- In the Kneipp system of therapy, ice-cold water
is sniffed up the nose as a way of training the
neuroautonomic regulation of the blood supply to
the nasal mucosa
85Traetment
- For intractable vasomotor rhinitis is surgical
reduction of the turbinates a septoplasty should
be performed.
86Atrophic Rhinitis
- Characterized by pronounced dryness of the nasal
mucosa. - Severe cases, especially with secondary bacterial
colonization, are marked by a fetid nasal odor
that is not perceived by the patient due to
degeneration of the olfactory epithelium.
87Etiology
- Primary atrophic rhinitis is unknown
- Secondary forms
- Extensive prior tumor resection
- Excessive use of nose drops drug abuse (cocaine)
- Previous radiotherapy for nasal and sinus tumors
88Treatment
- Conservative
- Symptomatic measures (saline nasal douche,
soothing mucosal ointments). - Surgery
- reduce the nasal cavity by the submucous
implantation of cartilage grafts.
89Hormonal Rhinitis
- Occurs mainly during pregnancy and is believed to
be caused by estrogen-induced swelling of the
mucosa with nasal airway obstruction.
90Rhinitis Medicamentosa
- This disease occurs mainly as a side effect from
the long-term use of decongestant nose drops
91Drug induced Rhinitis
- Antihypertensive drugs
- Beta-blockers,
- Angiotensin-converting enzyme (ACE) inhibitors
- Oral contraceptive
- Clinical
- symptoms consist of obstructed nasal
breathing, dry mucosa, and occasional olfactory
disturbances.
92Chronic Sinusitis
- Intranasal anatomic changes such as
- Septal deviation
- Septal spurs
- Chronic inflammation
- Allergy
- Trauma
- Neoplasms
- The common pathogenic mechanism is impaired
ventilation of the ostiomeatal unit
93- Chronic sinusitis frequently affects the
maxillary sinus and ethmoid cells
94Symptoms
- Pain (from feeling of pressure to persistent or
recurrent headaches) - Nasopharyngeal drainage (postnasal drip)
- Obstructed nasal breathing
95Diagnosis
- Rhinoscopy
- Endoscopy
- Imaging studies
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97Treatment
- Conservative treatment options
- Appropriate antiallergic therapy
- Sinus surgery
98Endoscopic sinus surgery
- The modern surgical treatment of chronic
sinusitis is performed intranasally under
endoscopic or microscopic control.
99Nasal Polyposis
- Genetic causes
- Chronic irritation of the mucosa, like that
occurring in chronic rhinitis or sinusitis - In response to allergic rhinitis and
acetylsalicylic acid (ASA) intolerance
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101Notice
- Nasal polyps are rarely observed in children.
- Most occur in a setting of cystic fibrosis.
102Symptoms
- Obstructed nasal breathing
- Hyposmia or anosmia
- Headache
- Snoring
- Rhinophonia clausa
- Frequent throat clearing
- Spread to the lower airways can lead to
laryngitis with hoarseness and bronchitic
symptoms.
103Diagnosis
- Rhinoscopic or endoscopic evaluation
- Computed tomography
- Allergy tests
- Olfactory testing
104Treatment
- Conservative measures
- Use of corticoid containing nasal sprays
- Systemic antihistamines
- Systemic steroids
- Surgical treatment
105Prognosis
- The prognosis is guarded even with modern
surgical techniques most meticulous ablative
sinus surgery cannot prevent a recurrence
106Mucoceles and Pyoceles
- Adhesions due to
- Postinflammatory
- Post-traumatic
- Postoperative
- The most common site of occurrence is the frontal
sinus, followed by the ethmoid cells, maxillary
sinus, and sphenoid sinus.
107Frontal Sinus
- Presents as an isolated, tense swelling over the
anterior wall of the frontal sinus - It may also cause inferolateral displacement of
the orbital contents
108Maxilla
- Swelling in the cheek area with upward
displacement of the orbital contents
109Ethmoid
- Proptosis, limited ocular movements, and diplopia
may also occur, depending on the location of the
mass.
110Diagnosis
111Treatment
- The treatment of choice is surgical removal of
the mucocele
112Rhinosinogenic Complications
113Orbital Complications
- They occur with highest frequency in children
under 6 years of age - Orbital edema
- Periosteitis
- Subperiosteal abscess
- Orbital cellulitis
- Orbital apex syndrome
- Cavernous sinus thrombosis
114Orbital Cellulitis
115Bone and Soft-Tissue Inflammations
116Osteitis and Osteomyelitis
- Osteomyelitis occurs mainly as a complication of
frontal sinusitis
117Symptoms
- The patient presents clinically with a tender,
doughy, erythematous swelling over the forehead
118Diagnosis
119Treatment
- The treatment of choice is surgical eradication
of the affected bone under antibiotic coverage
120Intracranial Complications
- Epidural, subdural and intracerebral abscesses
- Meningitis
- Sinus Thrombosis and Thrombophlebitis
121Question
- What is so serious regarding nasal foliculitis?
- Name the common symptoms of sinusitis.
- When orbit shift to the inferolateral the mucocel
perhaps is located in . sinus. - Name the causes of sinonasal polyposis.
- Subdural abscess is more common when the ..
Sinus is involved.