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Eye Movement Desensitization and Reprocessing

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Title: Eye Movement Desensitization and Reprocessing


1
Eye Movement Desensitization and Reprocessing
  • As a Tool for Treatment of Trauma AmongNative
    Americans
  • Mary (Mimi) Y. Sa, Psy.D.
  • MS Psychopharm
  • Taos-Picuris Service Unit
  • Indian Health Service
  • Mary.sa_at_ihs.gov

2
Unique Properties of Trauma Endured by Native
Americans
  • Generational in nature
  • Caused by fundamental destruction of major areas
  • - Family
  • - Culture
  • - Language
  • - Identity
  • - Introduction of alcohol
  • - Introduction of sexual abuse

3
History of Trauma Theories
  • Janet
  • Fixated on trauma
  • Unable to focus on the present
  • Freud
  • Considered the fixated trauma to be biologically
    based
  • Pavlov
  • Defensive reaction
  • US to CR, then CS to CR
  • Abraham Kardiner
  • Coined phrase posttaumatic stress
  • People with traumatic neurosis have enduring
    vigilance to environment

4
Physiology of Trauma
  • Profound alterations in stress hormones
  • Intense stress causes release of cortisol,
    epinephrine, norepinephrine, vasopressin,
    oxytocin and endogenous opioids
  • Norepinephrine
  • Vietnam veterans found to have higher levels of
    NE in their urine
  • Other studies have shown low urinary cortisol
    levels in Vietnam vets (which is negative
    feedback of NE)

5
Physiology of Trauma
  • 5HT
  • Inescapably shocked animals displayed decreased
    CNS serotonin levels (van der Kolk, 1985)
  • Decreased serotonin levels associated with
    impulsivity and aggression as well as emotional
    hyperarousal
  • Endogenous opioids
  • Stress related to analgesia
  • Fear associated with release of endogenous
    opioids
  • Re-exposure to stimuli similar to original will
    cause opioid release (even two decades later)
  • Release equivalent to 8mgs of morphine
  • Memory is impaired by release of ne and opioids

6
Symptomatology of PTSD
  • 3 most significant factors for developing
    disorder
  • Magnitude of exposure
  • Prior trauma
  • Social support
  • Abnormal physiological responses
  • Response to specific reminders of trauma
  • Response to intense, but neutral stimuli
    (startle)

7
Symptomatology of PTSD
  • Peter Lang
  • Associative networks
  • Where if one element of the memory reoccurs then
    the entire traumatic experience is relived
  • So certain sensations or emotions related to the
    trauma keep reoccurring and do not fade over time
  • Kolb
  • First to suggest trauma may cause permanent
    neuronal changes
  • Those changes can effect learning and stimulus
    discrimination

8
Symptomatology of PTSD
  • Abnormal startle response (ASR)-hallmark of PTSD
  • Studies show 93 of those with PTSD have this
  • Even after their symptoms remit
  • Versus 22 of normal controls
  • People with PTSD have inability to discriminate
    innocuous stimuli and continue to relive the
    threatening stimuli

9
Symptomatology of PTSD
  • Bimodal response
  • Hypermnesia, hyper-reactivity to stimuli and
    traumatic re-experiencing
  • Psychic numbing, avoidance, amnesia and anhedonia
  • In apparent attempt to counteract hyperarousal,
    traumatized people seem to shut down
  • They often go from stimulus to response without
    assessing the meaning
  • That makes them prone to freeze or overreact and
    intimidate others in response to minor
    provocations

10
Arousal and Traumatic Memories
  • High states of arousal may cause person to feel
    traumatized again
  • Therefore drugs that stimulate arousal
    (yohimbine, lactate) can cause flashbacks or
    panic attacks
  • When traumatized person is under stress, will
    secrete stress hormones (NE, endorphins,
    oxytocin) that interfere with memory consolidation

11
Arousal and Traumatic Memories
  • Oxytocin may be protective amnesic to help
    mothers forget the experience of childbirth.
  • In traumatized people, they will over remember
    the trauma any time they are physically aroused.

12
Memory, Trauma And Limbic System
  • Amygdala
  • Assigns meaning to emotional stimuli
  • Hippocampus
  • Is suppressed by stress hormones
  • Associated with memory and learning
  • Decreased hippocampal functioning causes
    behavioral disinhibition
  • In animals, stimulation of amygdala suppresses
    hippocampus

13
Traumatic memories
  • Include sensory memories (implicit vs. verbal)
  • These are subcortical systems and lie underneath
    the evaluative processes of the prefrontal cortex
  • So are outside of conscious awareness
  • Have no linguistic representation
  • Implicit memory can interfere with frontal lobe
    activity (executive function) and language
    production (Brocas area)

14
Traumatic Memories
  • So traumatic memory can be irrational
  • In traumatized person, the memory is fragmented
    into pieces
  • Non-traumatized people do not have a large
    physiological reaction to most memories that
    include sights, sounds, smells
  • When traumatized person has a memory, they live
    it
  • Their body re-experiences what it did at the time
    of the trauma
  • May not have language to describe it

15
Intense Arousal
  • When it is too great, individual often too
    overwhelmed to analyze and process sensory input
  • So tends to shut down or panic
  • Often people self-soothe with drugs or alcohol
  • Or increase pain by self-mutilation or anorexia
  • Arousal stays intense because the stimuli are not
    properly assimilated into the neural network

16
Narrow Interpretation of Stimuli
  • Often individuals with PTSD will interpret
    innocuous stimuli as traumatic as their ability
    to attach sensory information to new meaning has
    become inflexible
  • Van der Kolk describes PTSD as a frozen sensory
    world
  • It is therefore the job of the therapist to open
    up alternative interpretations of sensory data

17
Trouble With Talk Therapy for Trauma
  • Trauma is often stored in right hemisphere (per
    pet scan research) or subcortical regions.
  • Therefore, verbal discussion of trauma not always
    possible.
  • Van der Kolk (1994) asserts that understanding
    the physiological arousal to trauma may not help
    a person stop the process.

18
Trouble With Talk Therapy for Trauma
  • Often individuals are unwilling to
    verbally/intentionally recall the painful event.
  • Which may account for large drop-out rates in
    this type of therapy.
  • Especially since talk therapy is often slow.
  • The task in working with PTSD individuals is to
    help them integrate their fractured memories,
    without too much re-traumatization.

19
EMDR
  • History
  • Francine Shapiro walking through park
  • Started experimenting on friends
  • Then on clients
  • Now research is pervasive

20
EMDR
  • Explanation
  • Information processing therapy
  • Assumes humans are physiologically based sensory
    processing systems
  • Trauma blocks the natural processing of sensory
    stimuli

21
EMDR
  • Explanation (cont.)
  • Which stops the creation of new neural networks
    to interpret stimuli.
  • Adaptive processing is thought to occur when
    targeted memory is linked with neutral cognitions
    and reduced physiological arousal.

22
EMDR
  • Ideal for trauma victim
  • Positive response can come as quickly as minutes
    after treatment.
  • Do not need to talk about trauma.
  • Do not need to ask for as much trust from client,
    which may be helpful with traumatized individual.

23
Proposed Mechanism of Action
  • Some theorists suggest the bilateral stimulation
    of the brain simulates REM sleep and that both
    actions cause the integration of sensory
    experiences into long term memory.

24
Case Studies
  • Veterans
  • Children
  • Blow down
  • The dental example
  • Sexual abuse
  • Loss of loved one
  • Eczema guy

25
The Nuts and Bolts
  • Seems to work best on discrete trauma.
  • Traumatic events (especially those from
    childhood) often cause us to create negative
    beliefs about ourselves.
  • Have person identify the positive belief they
    want to have about themselves.
  • Identify level of distress
  • Have them rate this

26
The Nuts and Bolts
  • Identify intensity of negative belief.
  • Decide which type of stimuli to use (tapping vs.
    eye movements)
  • Stimulate for about one minute, then check in
  • Follow persons breathing, affect

27
The Nuts and Bolts
  • Go slowly or stop if person seems overwhelmed.
  • Keep going if person seems engaged but tolerant
    of affect.
  • Have person continue to rate distress level until
    distress level is low and negative belief seems
    untrue.
  • Now continue until positive belief is installed.

28
Personal Practices
  • My preferred method is tapping.
  • Client can close her eyes.
  • Some complain of dizziness with eye movement.
  • Be careful, however, about issue of touch
  • Make sure person is prepared/willing to go on
    huge emotional journey for the next 30 minutes or
    so.
  • i.e., make sure they dont have an important
    board meeting afterward
  • Give thorough explanation of the theory.
  • Then give thorough explanation of what the
    process entails.

29
Personal Practices
  • Ask permission to sit in front of client.
  • Do rating and identification process.
  • Begin tapping for about one minute.
  • Follow their breathing and affect to decide how
    long.

30
Personal Practices
  • Keep up process until person appears to have
    dramatically reduced their emotion and their body
    appears calm.
  • Finish with a guided imagery scene while you are
    tapping.
  • Instruct patient to eat well, hydrate and go to
    bed early as they will likely start to yawn and
    appear very fatigued.
  • Some therapists assert that it is like doing 20
    years of therapy in 20 minutes.

31
Use in Primary Care Setting
  • Mental health professional can be brought in on
    emergent basis to calm distressed individuals in
    hospitals in clinics.
  • Can help with anxiety about medical procedures or
    new diagnoses.
  • Can help if client discloses profound trauma in
    medical setting and client is in need of
    immediate relief.

32
Ways of Integrating Native Traditional
Beliefs/Practices
  • Many people report feeling a transfer energy.
  • Discussion of clients seeing sprits of their
    deceased loved ones after the grief was resolved.
  • If provider follows traditional practices, can
    utilize while tapping.

33
Provides a Unique Opportunity for Healing for
Natives
  • Because they can enjoy immediate relief, which
    will require less trust of the provider.
  • They do not have to discuss the trauma, which
    will possibly improve their willingness to try.

34
EMDR Certification
  • EMDR.com
  • Weekend 1 and weekend 2 training
  • Certification
  • Must be licensed mental health professional

35
Bibliography
  • Freud, S. (1919/1954). Introduction to
    Psychoanalysis and Neuroses. Standard Edition
    17 207-210. Translated and edited by Strachey.
    London Hogarth Press.
  • Janet, P. (1889). LAutomatisme Psychologique.
  • Kardiner, A. (1941). The Traumatic Neuroses of
    War. New York Hoeber.

36
Bibliography
  • Kolb, L.C., Multipassi, L.R. (1982). The
    conditioned emotional response A subclass of
    chronic and delayed post traumatic stress
    disorder. Psychiatric Annals, 12, 979-987.
  • Lang, P.J. (1979). A bio-informational theory of
    emotional imagery. Psychophysiology, 16,
    495-512.
  • Pavlov, I.P. (1926). Conditioned Reflexes An
    Investigation of the Physiological Activity of
    the Cerebral Cortex. Edited and translated by GV
    Anrep. New York Dover Publications.

37
Bibliography
  • Van der Kolk, B. (1994). The body keeps the
    score memory the evolving psychobiology of
    post traumatic stress. Harvard Review of
    Psychiatry, 1(5), 253-265.
  • Van der Kolk, B. van der Hart, O. (1991). The
    intrusive past The flexibility of memory and
    the engraving of trauma. American Imago, 48,
    425-454.
  • Van der Kolk, B.A., Greenberg, M.S., Boyd, J. and
    Krystak, J.H. (1985). Inescapable shock,
    neurotransmitters and addiction to trauma
    Towards a psychobiology of post traumatic
    stress. Biol Psychiatry, 20, 314-325.

38
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