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Pressure Ulcer Prevention and Management

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Title: Pressure Ulcer Prevention and Management


1
Pressure Ulcer Prevention and Management
  • Dipti Jethani RN BSN
  • Alverno College

All motion clips/images not labeled obtained
from Microsoft Clip Art
2
Navigation
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  • HOME /TABLE OF CONTENTS
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3
Table of Contents
4
Learning Objectives
  • Learner will be able to identify the stages of
    pressure ulcers
  • Learner will be able to identify patients at risk
    for pressure ulcers
  • Learner will be able to identify 3 ways to
    decrease risk and incidence of pressure ulcers
  • Learner will be able to identify 3 complications
    of pressure ulcers

5
Lets Start With A Case Study
  • Grace is an 84 year old female who was recently
    admitted into the hospital with a diagnosis of
    Pneumonia.
  • She has been weak, she used a cane before
    admittance in the hospital. She now is only
    mobile per wheel chair.
  • She also is an uncontrolled Type II Diabetic
    (Non-Insulin Dependent)
  • HOW WILL YOU PREVENT HER FROM DEVELOPING PRESSURE
    ULCERS?

Doheny, Patrick. (2007). Happy planet one.
Photograph. Retrieved from http//www.flickr.com
/photos/14132971_at_N05/1449122304/.
6
What Factor Most Puts Grace at Risk for Pressure
Ulcers?
  • Age
  • Lack of Mobility
  • Diabetes
  • Having Pneumonia
  • Having a cane shell beat the nurses up!

Click Here To Read Case Study Narrative
7
THAT S RIGHT
8
SORRY TRY AGAIN
Although this is a good answer, there is another
answer more fitting than this one, try again.
9
THE SKIN
  • Three Layers
  • Epidermis
  • Outermost Layer
  • Contains sensory receptors for pain, temp, touch,
    vibration, and pressure detection
  • Barrier to preserve moisture, vitamins, minerals,
    and proteins
  • Dermis
  • Lies beneath the epidermis, deeper and thicker
    too
  • Contains connective tissue, sebaceous glands
  • Contains fat sweat glands
  • Subcutaneous Tissue
  • Layer of fat and connective tissue
  • Layer of insulation to conserve body heat

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Lippincott Williams and Wilkins, 2011
10
Lets Review What are pressure ulcers??
  • Also known as Pressure/Bed Sores
  • Areas of cellular necrosis and skin breakdown
    most common over bony prominences
  • Can occur anywhere on the body
  • Most Common Sites Sacrum, Heels

http//www.nlm.nih.gov/medlineplus/ency/imagepages
/19091.htm For Educational Purposes, NIH
Lippincott Williams and Wilkins, 2011
11
Incidence of Pressure Ulcers
Anders, 2010 Sage Products Inc (2003)
Mereck Manual of Geriatrics, 2000 Krasner
(2008).
12
Risk Factors
  • Extrinsic Factors
  • Pressure
  • Friction
  • Shearing
  • Intrinsic Factors
  • Immobility
  • Inactivity
  • Incontinence
  • Malnutrition
  • Age
  • Mental Status

Mereck Manual of Geriatrics, 2000
13
Assessing the Risk
  • BEGINS ON ADMISSION
  • Skin Exam
  • On admission and every shift
  • History of Pressure ulcers
  • Recent weight loss
  • Mobility Status
  • Urinary/Bowel Incontinence
  • Dietary Intake/Nutr Status
  • Use Scales For Assessment
  • Braden Scale
  • Norton Scale
  • PUSH Tool

Guren, D., 2010.
14
Braden Scale
Cassell, 2009.
15
http//www.bradenscale.com/images/bradenscale.pdf
16
Prevention
  • BEGINS AT FIRST CONTACT
  • Turn patient at least every 2 hours
  • Do not place pts in a 90 degree lateral position
  • Puts more pressure on greater trochanter and
    lateral malleolus
  • Dont elevate HOB gt 30 degrees (except when
    eating) to minimize shearing forces
  • Avoid Fluorescent Light, it casts a blue tint to
    skin (Capezuti, 2008)
  • Check skin of high risk patients for changes in
  • Color, turgor, temp, and sensation.

Mereck Manual of Geriatrics, 2000
17
Prevention Cont.
  • Patient should not sit more than 2 hours
  • Sitting position puts increased pressure on
    ischial tuberosities.
  • Reposition patient every hour in chair
  • Teach patient to shift weight every 15 minutes
  • Do not use pillows/ rubber doughnuts
  • Keep skin surface clean and dry (Meticulous skin
    care)
  • As few pads as possible should be used
  • Main Points
  • Keep pressure off the area of breakdown
  • Clean and dress the wound
  • Maintain good nutrition

Mereck Manual of Geriatrics, 2000
18
Prevention
  • Monitor Lab Values
  • HgB lt12
  • Total Lymphocyte Count lt1200
  • Serum Albumin lt3.5
  • Serum Transferrin lt170
  • Promote Movement and Freq Position Changes

(Anders, 2010)
Mereck Manual of Geriatrics, 2000
19
Pressure Relief Aids
Lippincott Williams and Wilkins, 2011
20
Pressure Points
  • Most Develop Over 5 locations
  • Sacral Area
  • Greater Trochanter
  • Ischial Tuberosity
  • Heels
  • Lateral Malleolus
  • 90 occur in lower body

Agency for Health Care Policy and Research (2008)
Lippincott Williams and Wilkins, 2011 Abrass,
2004
21
Case Study
  • Grace has a history of COPD and has smoked 1 PPD
    for 22 years, but quit 7 years ago.
  • Her Diabetes has progressed and due to
    complications her Left foot was amputated.
  • On the last shift a small Stage I pressure ulcer
    was also discovered.

22
What complication should the nurse most focus on
preventing?
  • A. Progression of the Ulcer to a new stage
  • B. Increased Length of Stay
  • C. Infection
  • D. Sepsis
  • E. All of the Above
  • F. A and B only

Click Here To Read Case Study Narrative
23
Stages of Pressure Ulcers
  • 6 Stages
  • Staged according to depth of damage

Bright Hub Inc, 2011. Public Domain Image
Mereck Manual of Geriatrics, 2000
24
Suspected Deep Tissue Injury
  • Maroon or purple intact skin or a blood filled
    blister
  • Cause shearing or pressure on the underlying
    soft tissue
  • Before discoloration occurs, the area may be
  • Painful
  • Mushy, firm, or boggy
  • Warmer or cooler as compared to other tissue

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004
25
Stage I
  • An area of intact skin that does not blanch and
    is usually over a bony prominence.
  • NON-BLANCHABLE
  • Darkly pigmented skin may not show blanching but
    its color may differ from the surrounding area.
  • The area may be painful, firm or soft, or warmer
    or cooler when compared to the surrounding
    tissue.

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004
26
Stage II
  • A superficial partial thickness wound
  • Presents as a shallow, open ulcer without slough
    and with a red and pink wound bed.
  • This term shouldnt be used to describe
  • Perineal dermatitis, maceration, tape burns, skin
    tears or excoriation .
  • Only use to describe
  • An abrasion, a blister, or a shallow crater that
    involves the epidermis and dermis.

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004
27
Stage III
  • A full-thickness wound with tissue loss.
  • The subcutaneous tissue may be visible but
    muscle, tendon, or bone is not exposed.
  • Slough may be present but it does not hide the
    depth of the tissue loss.
  • Undermining and tunneling may be present.
  • Bone/Tendon are NOT visible

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004 NPUAP, 2007
28
Stage IV
  • Involves Full-Thickness skin loss
  • Can visibly see exposed muscle, bone, or tendon
  • Eschar and sloughing may be present as well as
    undermining and tunneling

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004
29
Unstageable
  • Involves full-thickness tissue loss.
  • The base of the ulcer is covered by
  • Slough yellow, tan, gray, green, or brown OR
  • Eschar tan, brown, or black
  • The pressure ulcer cannot be staged until enough
    eschar or slough is removed to expose the base of
    the wound

National Pressure Ulcer Advisory Panel (2007).
For Educational Purposes.
Abrass, 2004
30
Definitions
  • Friction Surface damage caused by skin rubbing
    against
  • another surface.
  • Shearing Trauma to skin caused by tissue layers
    sliding against each other, results in disruption
    of blood vessels.
  • Maceration Softening of tissue by soaking in
    fluids.
  • Debridement Removal of damaged tissue.
  • Eschar Thick, leathery necrotic tissue damaged
    tissue.
  • Slough Loose, stringy necrotic tissue
  • Undermining Tissue destruction underlying intact
    skin
  • along wound edges.
  • Tunneling A narrow channel/passageway extending
    into
  • healthy tissue.

Oklahoma Foundation for Medical Quality, 2009
31
Case Study Cont.
  • Which of the following is Grace most at risk for?
  • Friction
  • Shearing
  • Maceration
  • Laceration

Click Here To Read Case Study Narrative
32
Case Study
Click Here To Read Case Study Narrative
  • Grace became incontinent of bowel and bladder.
  • She has a decreased appetite and has become more
    confused since the last shift.
  • A urine specimen was obtained and results show
    she has a UTI.

33
What measures can you take to prevent progression
and development of pressure ulcers?
34
Pathophysiology
  • Affected area becomes hypoxic and ischemic d/t
    press exerted on it
  • ?
  • Decreased blood flow to site
  • ?
  • Capillaries Collapse, Thrombosis occurs
  • ?
  • Tissue Edema/Necrosis
  • ?
  • Accumulation of waste products at site
  • ?
  • Tissue Breakdown
  • ?
  • Cell Death

Lippincott Williams and Wilkins, 2011
35
Stress Response and Pressure Ulcers
  • Stress occurs
  • ?
  • Adrenal Glands produce Epi
  • ?
  • SNS releases NE
  • ?
  • NE causes peripheral vasoconstriction
  • ?
  • Decreased Oxygen Delivery
  • ?
  • Epi enhances production of chalone
  • ?
  • Chalone protein depresses regeneration of
    epidermal tissue
  • ?
  • Tissue Breakdown
  • ?
  • Cell Death

Epi Epinephrine NE Norepinephrine
Place mouse over chalone to see definition
Maklebust, J., Sieggreen, M. (2001).
36
Aging
  • Muscle fat are lost with aging (to spread out
    press)
  • Skin Elasticity Decreases
  • ? in ascorbic acid levels
  • BVs Connective tx more fragile
  • Lowers threshold of pressure injury
  • ? in of Dermal BVs
  • Incr risk of ischemic injury by press and
    shearing forces
  • Wound healing ?ed
  • Repair rate declines
  • Cell proliferation ?es
  • Wound tensile strength ?es
  • Collagen deposition ?es

Lippincott Williams and Wilkins, 2011 Mereck
Manual of Geriatrics, 2000
37
Inflammation
  • Damaged BVs
  • ?
  • Exposed Collagen
  • ?
  • With Thrombin exposed collagen stimulates
    platelet activity
  • ?
  • Activation, aggregation, and adhesion of
    platelets and release mediators
  • ?
  • Stimulates Vasoactive substances
  • ?
  • Breakdown products attract nuetrophils and
    macrophages
  • ?
  • Monocytes become Macrophages
  • ?
  • Release growth factors
  • ?
  • Trigger Fibroblasts to secrete collagen
    proteins
  • ?
  • Wound becomes beefy red and bleeds

Hulse, 2011
38
Genetics
  • Study by Bregstorm Braden (2002)
  • found A.A. lowest risk for Stage I ulcers
    Caucasians at highest risk for developing Stage I
    ulcer
  • For stages II-IV AA most at risk for progression
    of an ulcer Caucasians least at risk
  • Certain Medical Conditions
  • Diabetes M.
  • Peripheral Vascular Dx
  • Predisposed to developing pressure sores

Capezuti, 2008
Mereck Manual of Geriatrics, 2000
39
Pressure Ulcers on Heels
  • Account for 20 of all pressure ulcers
  • Easy to acquire hard to heal
  • Pressure relief
  • Pillows (floating)
  • Heel Protector Boots
  • Dressing if necessary
  • Foam Pads

Guren, D., 2010.
40
Complications
  • Progression of pressure ulcer
  • Secondary Infections
  • Ex. Sepsis, Cellulitis
  • Osteomyelitis ?Loss of limb from bone involvement
  • Marjolins Ulcer
  • Squamous cell carcinoma within the ulcer
  • Increased Length of Stay
  • Increased Costs
  • Death

Lippincott Williams and Wilkins, 2011
Mereck Manual of Geriatrics, 2000 Capezuti, 2008
41
Treatment
  • Prevention Strategies
  • Pressure Reduction
  • Avoiding Friction/ Shearing Forces
  • Stage III/IV
  • Debridement of necrotic tissue
  • Freq Irrigation (2-3x/d)
  • Dressing of the wound
  • Encourage movement

Abrass, 2004 Anders, 2010
42
POP QUIZ
  • Identify the pressure ulcer stage of the
    following area of skin abnormality .

Public Domain Image obtained from
http//i.ytimg.com/vi/QvcjH98ipeU/2.jpg
43
  • IF YOU SAID
  • STAGE III
  • YOU WERE RIGHT!!

44
Nutrition
  • Need adequate intake for wound healing and immune
    response
  • Incr protein/caloric intake
  • Supplement with multi-vitamins (A C)
  • Consult with a Dietician
  • Loss of gt 15 of lean body mass interferes with
    wound healing
  • Immune Function Loss
  • gt Incr risk of infection and decr wound healing

Anders, 2010 Maklebust, J., 2001
45
Nursing Implications
Krasner, 2008
46
Documentation
  • Location
  • Size
  • Dressing
  • Type of irrigation soln, drsg applied
  • Drainage
  • Amt, color, odor
  • Undermining/Tunneling Present
  • Infection s/s
  • Character of wound
  • Presence of slough, granulation tissue, etc
  • Pressure relieving measures used

Hill Rom Services Inc., 2007
47
CASE STUDY CONT.
  • What aspect of Graces existing condition is the
    most influencing factor for increasing her risk
    for pressure ulcers?
  • Pneumonia/COPD
  • Diabetes
  • Venous Insufficiency
  • Lack of Mobility
  • None of the Above
  • All of the Above

Click Here To Read Case Study Narrative
48
Case Study Conclusions
  • Press ulcers can increase morbidity and risk for
    complications
  • Grace represents a typical patient
  • Documentation and thorough assessment is a
    necessity
  • FOR A VIRTUAL TOUR OF SKIN BREAKDOWN SEE NEXT
    SLIDE

Doheny, Patrick. (2007). Happy planet one.
Photograph. Retrieved from http//www.flickr.com
/photos/14132971_at_N05/ 1449122304/.
49
http//www.youtube.com/watch?vEyuguc7KKC4feature
player_embeddedat61 (Click here to see video)
50
Did you meet the objectives??
  • Identify the stages of pressure ulcers
  • Identify patients at risk for pressure ulcers
  • Identify 3 ways to decrease risk and incidence of
    pressure ulcers (prevention)
  • Identify 3 complications of pressure ulcers

Click the link if you want to review!
51
TRY AND RUN WITH IT
Questions? Email jethandr_at_alverno.edu
52
References
  • Abrass, I., Kane, R., Ouslander, J. (2004).
    Essentials of Clinical Geriatrics. (5th ed.).
    McGraw Hill-Companies, Inc.Hightstown, NJ.
  • Agency for Health Care Policy and Research
    (2008). AHCPR Supported Guide and Guidelines
    Internet. Rockville MD. Retrieved on April 2,
    2011 from http//www.ncbi.nlm.nih.gov/books/NBK17
    977/.
  • Anders, J., Heinemann, A., Leffmann, C.,
    Leutenegger, M., Profener, F.,
    Von-Rentein-Kruse, W. (2010). Decubitus Ulcers
    Pathophysiology and Primary Prevention.
    Deutsches Arzteblatt International, 107 (21)
    371-82.
  • Aurora Health Care (2010). Skin Integrity
    Alterations Potential/Actual for Adult
    Inpatients. Milwaukee, WI Aurora HealthCare.
  • Bright Hub Inc. (2011). Healing Bedsores.
    Retrieved on April 2, 2011 from http//www.bright
    hub.com/health/alternativemedicine/articles/52007.
    aspx.
  • Capezuti, E., Fulmer, T., Mezey, M., Zwicker,
    D. (2003). Evidenced Based Geriatric Nursing
    Protocols For Best Practice. (3rd ed). Springer
    Publishing Co., New York, NY.
  • Cassell, C. (2009). Pressure Ulcer Assessment
    The Braden Scale for Predicting Pressure Ulcer
    Sore Risk. Health Services Advisory Group.
  • Guren, D. (2010). Skin is in positioning your
    surgical patient matters. Retrieved March 28,
    2011 from http//uwcne.net/grandrounds/display.asp
    ?ID48 submitVideo.

53
References Cont
  • Hill-Rom Services Inc. (2007). Guidelines for
    staging of pressure ulcers. Brochure.
  • Hulse, J. (2011). Skin and Wound Care. Pesi
    Health Care. Confrence.
  • Krasner, D., McNeil, M., Weir, D. (2008). The
    Pressures On Getting it Right on Admission.
    Norcross, GA Molnlycke Health Care.
  • Lippincott. (2011). Professional Guide to
    Pathophysiology (3rd ed.). Lippincott Williams
    and Wilkins. Ambler, PA.
  • Maklebust, J., Sieggreen, M. (2001). Pressure
    Ulcers Guidelines for Prevention and
    Management. (3rd ed.). Ambler, PA. Lipponcott
    Williams and Wilkins.
  • Molnlycke Health Care (2007). Mepilex Border
    Sacrum. Brochure. Norcross, GA.
  • National Pressure Ulcer Advisory Panel. (2007).
    Pressure Ulcer Category/Staging Illustrations.
    Retrieved on April 1, 2011 from
    http//www.npuap.org/resources.htm.
  • Nucleus Medical Media. (2011). Pressure Sores
    youtube video Retreived from http//www.youtube.
    com/watch?vEyuguc7KKC4featureplayer_
    embeddedat61.

54
References Cont.
  • Oklahoma Foundation for Medical Quality. (2009 ).
    Appendix A Glossary Pressure Ulcer Terms.
    Retrieved on April 10, 2011 from http//
    www.ofmq.com/Websites/ofmq/Images/SOS20PU20Tool
    kit/Appe ndix20A.pdf .
  • Porth, C., Matfin, G. (2009). Pathophysiology
    Concepts of Altered Health States (p. 38-46).
    Philadelphia, PA Lippincott Williams Wilkins.
  • Sage Products Inc. (2003). What the experts say
    about the financial implications of pressure
    ulcers. Brochure. Cary, Il.
  • The Merck Manual of Geriatrics 3rd Edition
    (2000), (pp. 1317-1322). Whitehouse Station, NJ
    Merck Research Laboratories.
  • US Dept of Health Human Services, National
    Institutes of Health (2010). Areas Where
    Bedsores Occur. Online Image. Retrieved on
    April 1, 2011 from http//www.nlm.nih.gov/medline
    plus/ency/imagepages /19091.htm.
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