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Asthma Treatment

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Asthma Treatment – PowerPoint PPT presentation

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Title: Asthma Treatment


1
Asthma Treatment
  • James Smith, D.O.
  • 11/24/2005

2
Chief Complaint and History
  • CC Patient come to the office c/o continued SOB,
    wheezing, and nocturnal cough.
  • HPI Patient is a known asthmatic who is on
    maximum treatment for chronic persistent asthma
    yet still has above complaints. He notes that
    symptoms are worse when he is out in the cold
    weather.

3
Physical Exam
  • Gen Mild respiratory distress with noted
    accessory muscle use.
  • Heart RRR. No murmurs
  • Resp Decreased air movement in all lung fields.
    Expiratory wheezes noted throughout.
  • MS Increased sub-occipital fullness. Increased
    cervical and scalene muscle tension. C4 F Rl Sl.
    Inhalation 1st rib on right. T2 F Sr Rr with
    right 2nd rib posterior.

4
  • MS (Cont.) Decreased overall compliance of
    inspiratory effort of rib cage. Increased
    diaphragm tension with some flattening. L3-4 E Sr
    Rr.
  • To further understand some of these findings lets
    review some of the corresponding anatomical
    relationships.

5
Functional Anatomy
  • C-spine OA, AA, C3-C5, and scalene muscles
  • T-spine and CV joints paraspinal sympathetic
    ganglia
  • Rib cage compliance and respiratory function
  • Thoracoabdominal diaphragm
  • L-spine compensation and relationship to
    diaphragm

6
C-spine OA , AA, C3-C5, and scalene muscles
  • Vagus nerve path through OA and AA
  • C3-C5 and Phrenic nerve origin
  • Scalene attachment to 1st and 2nd ribs

7
T-spine and CV joints paraspinal sympathetic
ganglia
  • Sympathetic chain gaglion
  • Vertebra and rib association
  • Posterior rib angles position for fulcrum

8
Rib cage compliance and respiratory function
  • Complex neuromuscular association
  • Cervical and scalene muscle attachments
  • Nerovascular complex and congestion

9
Thoracoabdominal diaphragm
  • Convex dome shape
  • Costal attachment of edges
  • Lumbar attachment of the crual muscles
  • Phrenic nerve inervation
  • Vascular/Lymphatic openings

10
L-spine compensation and relationship to
diaphragm
  • Crual muscle origin and attachment to lumbar
    spine
  • Lumbar spine fascia and integration with
    diaphragm fascia

11
Expected Results
  • Normalizing Parasympathetic/sympathetic tone
  • Increase of the following
  • Acute burst of catecholamines and bronchial
    dilation
  • Thinning of my secretions
  • Decrease of the following
  • Goblet cell hyperplasia over the long term
  • Smooth muscle hyperplasia over the long term
  • Quantity of mucus production

12
  • Improving diaphragmatic motion
  • Allowing for increased tidal volume
  • Improving lymphatic flow and decreasing vascular
    congestion
  • Improving thoracic cage compliance
  • Increases AP diameter and overall respiratory
    excursion
  • Decreases muscle and fascial tension allowing for
    neurovascular imrpovement

13
  • Improve/Normalize vagus and phrenic nerve
    involvement.
  • Decreases mucus production and bronchial spasm
  • Improves neural stimulation of the diaphragm
  • Correct lumbar spine compensations
  • Decreases tension into diaphragm via crual
    muscles
  • Maintain natural lumbar lordosis improving
    thoracoabdominal mechanics for respiration

14
Possible Treatments
  • S-O release, OA and AA treatment
  • ME for C3-C5 dysfunction
  • Soft tissue stretching of scalene muscles
  • Correction of 1st rib dysfunction
  • Asthma reflex at T2 on left (if rib involved
    inhibitory pressure works well)

15
Possible Treatments
  • Rib raising (seated or supine)
  • Increase thoracic cage compliance
  • Abdominal diaphragm redoming
  • HVLA for lumbar compensation

16
S-O release
  • Patient supine
  • Place finger tips at base
  • of occiput
  • Apply anterior pressure
  • Allow head to extend over fingers stretching the
    diaphragm
  • Ask patient to take several deep breaths in and
    out
  • Repeat monitoring for a release

17
Soft tissue stretching of paraspinal and
scalene muscles
  • Cross hands allowing head to rest on them
  • Flex head and have patient push head back into
    them
  • Apply one hand to head and other to 1st and 2nd
    rib area.
  • Sidebend head away and have patient push back
    towards shoulder

18
OA and AA treatment
  • Patient supine
  • Evaluate for rotation and sidebending
    restrictions
  • Take patient to restriction barriers, flex head
    to 40 degrees
  • Ask patient to turn back to ease
  • Have patient relax and take patient to a new
    restrictive barrier
  • Repeat series 3 times monitoring for release

19
ME for C3-C5 dysfunction
  • Patient supine
  • Like AA and OA take patient to restrictive
    barrier
  • Do not flex head to 40 degrees
  • Ask patient to turn head back into ease
  • Have patient relax and take to new restrictive
    barrier
  • Repeat cycle 3 times monitoring for release

20
Correction of 1st rib dysfunction
  • Apply direct contact
  • to 1st rib head
  • May apply to one
  • rib or both at the
  • same time
  • Apply direct pressure
  • caudad and ask patient to shrug shoulders
    upwards
  • During this have patient take a deep breath in
  • Repeat upto 3 times or until release is felt

21
Rib raising (supine)
  • Patient supine
  • Contact posterior
  • rib angles with fingertips
  • Apply anterior
  • pressure with lateral traction
  • Continue and monitor for muscle tension release

22
Abdominal diaphragm redoming
  • Patient is supine
  • Place hands at the base of the ribs
  • Place thumbs under costal margin
  • Have patient take a breath in and resist
  • Have patient exhale and follow diaphragm up
  • Keep thumbs inplace and have patient take another
    breath

23
HVLA for lumbar compensation
  • Lay patient in left lateral recumbent position
  • Extend or flex from below to level of dysfunction
  • Rotate and sidebend patient from above down to
    level of dysfunction
  • Apply rotational thrust through innominant in a
    anterior and inferior motion
  • Recheck when finished
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