Title: NURSING MANAGEMENT OF PATIENTS WITH ALTERATIONS IN CARDIOVASCULAR FUNCTIONING
1NURSING MANAGEMENT OF PATIENTS WITH ALTERATIONS
IN CARDIOVASCULAR FUNCTIONING
- DEBORAH PEPPER HOOVER, RN, MSN
2OXYGENATION
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4Blood Flow In the Heart
- VENOUS SYSTEM
- RIGHT ATRIUM
- TRICUSPID VALVE
- RIGHT VENTRICLE
- PULMONIC VALVE
- PULMONARY ARTERY
- LUNGS
- PULMONARY VEINS
- LEFT ATRIUM
- MITRAL VALVE
- LEFT VENTRICLE
- AORTIC VALVE
- AORTA
- BODY
5See page 533
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7See page 534
8CORONARY CIRCULATION
- RIGHT CORONARY ARTERY
- LEFT MAIN CORONARY ARTERY LEFT ANTERIOR
DESCENDING LEFT CIRCUMFLEX
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10See page 534
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12See page 536
13Cardiac Output HR x SV
- THE ABSOLUTE BOTTOM LINE FOR ASSESSMENT IS
CARDIAC OUTPUT!!!!!
14Stroke Volume - amt. of blood pumped per beat
- PRELOAD - stretch on the heart before
contraction Frank-Starling Law of the heart - CONTRACTILITY - forcefulness of contraction of
ventricular fibers Positive INOTROPIC
- increase - AFTERLOAD - peripheral resistance against which
the left ventricle must pump (press. that must be
exceeded)
15Sympathetic Stimulation of the Heart
- Mediated by Beta adrenergic receptors
- Releases norepinephrine
- Causes 1. Inc. heart rate. 2. Inc. in speed of
impule conduction in the AV node 3. Inc. in
force of atrial ventricular contraction
16Parasympathetic Stimulation of the Heart
- Mediated through the vagus (X) nerve
- Releases acetylcholine
- Decreases heart rate
- Decreases speed of conduction through AV node
17Adrengeric Receptors in the Blood Vessels
- ALPHA STIMULATION- causes vasoconstriction
- BETA STIMULATION- causes vasodilitation
18HISTORICAL DATA
- Question about hx of chest pain, shortness of
breath ( esp. paroxysmal nocturnal dyspnea ),
alcoholism, anemia, rheumatic fever, congenital
ht. dz., stroke, HTN, thrombophlebitis,
intermittent claudication, varicosities, edema - ? Risk factors - Inc. Lipids ? Smoking
(see page 544 for risk factors)
19Inspection Palpation of the C-V System
- Overall view of skin color, hair distribution,
edema, abnormal pulsations - Gauge capillary filling time (within 3 sec.)
- Palpate pulses for normal, bounding, thready, or
absent (0-3) - ? Thrill ? Homans sign ? Bruit
20Assessing Jugular Venous Pressure (see page 547)
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22Inspection Palpation of the Thorax for Cardiac
Status
- Inspection for abnormal thoracic structures
vibrations - Palpate auscultatory areas for thrills
- Note any heaves
- Locate the PMI
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24Auscultation of the Heart
- Listen in the auscultatory areas starting with
diaphragm then bell - Concentrate on identifying S1, S2, systole
versus diastole - Feel for carotid pulse to determine first heart
sound (S1) or lub - Check for pulse deficit by listening to apical
impulse while counting radial pulse
25HEART SOUNDS
- LUB or S1 - ASSOCIATED WITH CLOSURE OF THE
TRICUSPID MITRAL VALVES - DUB or S2 - ASSOCIATED WITH CLOSURE OF THE
PULMONIC AORTIC VALVES
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29Auscultation
- Third heart sound (S3) - extra, heard in early
diastole (rapid filling of ventricle), associated
with left ventricular failure - Fourth heart sound (S4) - extra, heard in late
diastole (atrial contraction), associated with
resistance to ventricular filling like
hypertension - Normal physiologic splitting of S2 - split at
pulmonic area with inspiration
30Gallops (see page 549)
31Murmurs
- Produced by turbulent blood flow
- Graded on a scale from I - VI I - soft,
faint VI - heard without a
stethoscope - Record timing, location, pitch, characteristics
- Associated with valve disorders or abnormal blood
flow patterns
32Diagnostic Studies
- EKG - 12 lead, telemetry, ambulatory (Holter),
treadmill exercise test (GXT) - Echocardiogram - ultrasound waves
- Nuclear cardiology - MUGA, Thallium imaging,
Technetium scan, PET scan - Serum enzymes - CK-MB fraction, myoglobin
- Cardiac catheterization (arteriography)
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34ECG or EKG (Electrocardiogram) Monitoring (see
page 553)
35Echocardiogram (see page 555)
36CORONARY ARTERY DISEASE
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39 Normal Atherosclerotic Artery (see page
594)
40CORONARY ARTERY DISEASE (CAD)
- Progressive obstruction of blood flow through one
or more coronary arteries - Disease progresses for many years before symptoms
develop - Nonmodifiable risk factors - AGE, SEX, FAMILY
HISTORY - Modifiable risk factors - SMOKING,
HYPERLIPIDEMIA, HTN, Diabetes, Obesity, Lack of
Exercise, Stress, The Pill
41Angina Pectoris - Choking of the Chest
- STABLE ANGINA PECTORIS - occurs when physical
exertion or emotional stress causes an increase
in myocardial O2 demand beyond the ability of
coronary arteries to increase blood supply - UNSTABLE ANGINA PECTORIS - coronary arteries are
unable to meet the O2 demands of the myocardium
at rest
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43Other Types of ANGINA
- INTRACTABLE - chest pain that is refractory to
interventions such as meds - PRINZMETALS OR VARIANT - severe, prolonged chest
pain in the absence of precipitating factors
thought to develop from coronary artery spasm - NOCTURNAL - chest pain that occurs only at night
- See page 597 SILENT ISCHEMIA
44Cardiac Markers
- CPK-MB 3-6hrs 12-24hrs 2-3dys Can rule out MI
Other disorders can elevate like trauma, surgery,
exercise - Myoglobin 1-2hrs 3-15hrs 20-24h Levels rise
quickly with MI other disorders can cause
elevation value ends 24 - Troponin T 2-4hrs 4-6hrs weeks Useful in ruling
out MI for up to one wk Angina uremia can
elevate - Troponin I 4-6hrs 14-18hrs 6-7dys Very cardiac
specific costly
45NITRATES
- Nitroglygerin is most common
- Side effects from vasodilation - headache,
hypotension, syncope - Need nitrate-free interval
- Sudden withdrawal may result in death
- Causes a generalized vasodilation throughout the
body - Decreases venous return (preload) BP
(afterload) thus decreasing the O2 demands of
heart
46INNOVATIONS IN CAD
- PERCUTANEOUS TRANSLUMINAL ANGIOPLASTY (PTCA)
- INTRACORONARY STENTS
- LASER ANGIOPLASTY
- ATHERECTOMY
- CORONARY ARTERY BYPASS GRAFT SURGERY (CABG)
47Percutaneous Transluminal Coronary Angioplasty
PTCA (see page 602)
48Intracoronary Artery Stent (see page 603)
49Three Coronary Artery Bypass Grafts (see page 604)
50NURSING DIAGNOSES
- PAIN RT MYOCARDIAL ISCHEMIA
- ANXIETY RT PAIN FEAR OF DEATH
- POTENTIAL DISTURBANCE IN SELF CONCEPT
- KNOWLEDGE DEFICIT ABOUT DISEASE PROCESS,
THERAPIES METHODS TO AVOID COMPLICATIONS