Title: Glaucoma "the silent thief of sight"
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2NANNM CONTINUING EDUCATION COMMITTEE FEDERAL
TEACHING HOSPITAL GOMBE
- TOPIC GLAUCOMA THE SILENT THIEF OF SIGHT.
- PRESENTER ABDULWAHAB USMAN (NOI)
- DATE12 TH APRIL,2016
- VENUE CONFERENCE HALL FTH GOMBE
3OUTLINE
- INTRODUCTION
- DEFINITION
- BRIEF ANATOMY AND PHYSIOLOGY
- CLASSIFICATION
- MANAGEMENT
- NURSING CARE
- CHALLENGES
- RECOMMENDATIONS
- CONCLUSION
- REFERENCES
4INTRODUCTION
- Glaucoma is considered to be the second largest
cause of blindness after cataract worldwide, and
the foremost cause of irreversible blindness. - In 2013, the number of people with glaucoma
worldwide was estimated to be 64.3 million ,
increasing to 76.0 million in 2020 and 111.8
million in 2040. - Glaucoma is mostly refer to as the silent thief
of sight due to its asymptomatic nature at early
stage. - Early detection and effective treatment are
necessary to prevent loss of vision.
5DEFINITION
- Glaucoma is a group of ocular disease
characterized by progressive optic neuropathy
resulting in optic disc defect and irreversible
visual field loss that are usually associated
with raised intraocular pressure.
6BRIEF ANATOMY PHYSIOLOGY
7PRODUCTION AND FLOW OF AQUEOUS HUMOUR
- Aqueous humour, derived from plasma, is produced
from ciliary process. From there, it passes to
posterior chamber and then around iris to
anterior chamber. From the anterior chamber, it
either drains into schlemn's canal via trabecular
meshwork Trabecular outflow or goes back into
ciliary process towards venous circulation
Uveoscleral outflow.
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10CLASSIFICATION
- Congenital /developmental Glaucoma
- Primary
- Secondary
11CONGENITAL GLAUCOMA
- Congenital glaucoma is further classified as
follows depending on the age of onset. - True congenital glaucoma (40)
- Infantile glaucoma (55)
- Juvenile glaucoma (5)
- It affects 1 in 10,000 birth of the population .
- 70 bilateral.
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13CLINICAL FEATURES
- Corneal oedema
- Lacrimation , photophobia and blepharospasm
(Classic Triad). - Corneal enlargement.
- Haabs striae.
- Raised IOP
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15EVALUATION
- Tonometry
- Measurement of corneal diameter by callipers
- Slit lamp examination
- Ophthalmoscopy
- Gonioscopy
16MANAGEMENT
- Primarily surgical.
- Goniotomy.
- Trabeculotomy if goniotomy fails or when the
corneal clouding prevent visualisation of the
angles. - Trabeculectomy.
17PRIMARY GLAUCOMA
- Primary open angle glaucoma (POAG)
- Primary angle closure glaucoma (PACG)
- PRIMARY OPEN ANGLE GLAUCOMA
- Adult onset
- An open angle of normal appearance.
- Most prevalent type of Glaucoma (1/3)
- 1 in 100 0f the population gt 40 years.
18RISK FACTORS
- Age gt 40 years
- Race develop earlier and more severe in black
than white - Family history
- Diabetes and Hypertension
- Myopes
- Low CCT lt 555um
- Retinal diseases
- Steroids
19CLINICAL FEATURES
- Asymptomatic at early stage
- Headache
- Eye ache
- Scotoma
- Difficulty in reading and close work
- Delayed dark adaptation
- Decrease visual acuity
- Increase IOP
- Cupping of the optic disc
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23EVALUATION
- Visual acuity
- Slit lamp examination
- Tonometry
- Pachymetry
- Perimetry
- Ophthalmoscopy
- Gonioscopy
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26MANAGEMENT
- Initial therapy is medical , with surgery as the
last resort. - Beta blockers eg Timolol maleate and Betaxolol
- Prostaglandin analogue eg Latanoprost.
- Adrenergic drugs eg Brimonidine
- Carbonic anhydrase inhibitors eg Dorzolamide and
Acetazolamide - Miotics eg pilocarpine
- Hyperosmotic agent eg mannitol
- SURGERY
- Laser trabeculoplasty
- Trabeculectomy
27NORMAL TENSION GLAUCOMA
- Also called low tension glaucoma, is when typical
glaucomatous changes and visual field defects are
associated with an IOP that is constantly below
21mmHg - It is believed to result from low vascular
perfusion which makes the optic nerve head more
susceptible to normal IOP. - Betaxolol is the drug of choice
28OCULAR HYPERTENSION
- Also called glaucoma suspect is when a patient
has an IOP constantly more than 21mmHg but no
optic disc and visual field defects. - The patient should be carefully monitored and
should be treated as a case of POAG in the
presence of risk factors. - Significant diurnal variation of more than 8mmHg
between the lowest and the highest values of IOP - IOP constantly more than 28mmHg
- Significant asymmetry in the cup size of the two
eyes i.e gt 0.2.
29PRIMARY ANGLE CLOSURE GLAUCOMA
- Primary angle closure disease is characterized by
apposition of the peripheral iris against the
trabecular meshwork, resulting in obstruction of
the aqueous outflow. - Primary angle closure glaucoma is used only when
the optic disc and visual field changes are
present.
30RISK FACTORS
- Sex More in female than male. 41.
- Hereditary
- Age More in advance age with highest frequency
in 6th and 7th decades. - Hypermetropics due to shallow anterior chamber
and short axial length. - Race More in South East Asians, Chinese and
Eskimos than blacks.
31CLINICAL FEATURES
- Severe pain
- Nausea , vomiting and prostration
- Red eye
- Photophobia
- Lacrimation
- Raised IOP (40-70mmHg)
- Ocular structures becomes oedematous
- Closed angle of anterior chamber
- Fixed and dilated pupils
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33MANAGEMENT
- Primary angle closure disease is a serious
ophthalmic emergency and needs to be manage
aggressively as follows - Immediate medical therapy to lower IOP
- Systemic hyperosmotic agents e.g Mannitol and
Glycerol - Systemic CAI e.g Acetazolamide 500mg t.d.s
- Topical anti-glaucoma drugs beta blockers and
prostaglandin analogues. - Analgesia
- Antiemetic
- Antinflammatory
34- Definitive therapy
- Surgeries i.e laser peripheral iridotomy, laser
iridotomy and trabeculectomy - iii. Prophylaxis of the fellow eye
- Prophylaxis surgery should be performed on the
fellow asymptomatic eye as early as possible as
chances of acute attack is 50 in such eye. - iv. Long term glaucoma surveillance and IOP
management in both eyes.
35SECONDARY GLAUCOMAS
- Depending on the cause, secondary glaucoma are
named as follows - Lens induced glaucoma
- Inflammatory glaucoma
- Neovascular glaucoma
- Glaucoma associated with intraocular haemorrhage
- Glaucoma associated with intraocular tumor
- Steroid induced glaucoma
- Traumatic glaucoma
- Glaucoma in aphakia
- Pseudoexfoliative glaucoma
- Pigmentary glaucoma
36NURSING CARE PLAN FOR A GLAUCOMA PATIENT
NURSING DIAGNOSIS NURSING OBJECTIVE NURSING INTERVENTION SCIENTIFIC RATIONALE EVALUATION
Disturbed visual sensory perception related to altered sensory reception as evidenced by loss of visual field/decreases visual acuity To maintain current visual field/acuity without further loss Determine type and degree of visual field loss Allow expression of feelings about loss and possibility of loss of vision Administer prescribed medications as indicated Prepare patient for surgery and care post operatively Baseline for evaluation Help reassure the patiient that vision loss cannot be restored but further loss can be prevented Control IOP and prevent further loss of vision To prevent further loss of vision Patient visual field /acuity was maintained without further loss
37NURSING DIAGNOSIS NURSING OBJECTIVE NURSING INTERVENTION SCIENTIFIC RATIONALE EVALUATION
Anxiety(actual) related to loss of vision as evidenced by negative self-talk Allay anxiety Determine level of anxiety Give accurate and honest information Encourage patient to express feelings and concerns. Identify helpful resources and people Baseline for evaluation Lessens anxiety related to unknown. Provide opportunity for patient to deal with reality of situation and clarify misconceptions. Provide reassurance that patient is not alone in dealing with problem Patient anxiety was allayed
38NURSING DIAGNOSIS NURSING OBJECTIVE NURSING INTERVENTION SCIENTIFIC RATIONALE EVALUATION
Knowledge deficit regarding disease process Patient will understand pathology and treatment of glaucoma Review pathology and lifelong need for treatment Demonstrate proper technique for administration of eye drops Discuss dietary consideration (adequate fluid and fiber intake) Identify potential side effects of treatment Provides opportunity to clarify and dispel misconception Enhance effectiveness of treatment To prevent constipation and straining during defecation May require medical evaluation and possible change in therapeutic regimen Patient understood pathology and treatment of Glaucoma
39CHALLENGES
- Late presentation
- Lack of awareness
- Delay in referral
- Lack of expertise
- Poor compliance with medical treatment
- Lack of equipments
- High cost of drugs
- Fake drugs
- Surgeons reluctance to offer surgery
40RECOMMENDATIONS
- Routine glaucoma screening to help detect the
disease at early stage and prevent further loss
of vision. - Increase awareness through health talk and media.
- Early referral of patient from other health
professionals to ophthalmic specialist. - Training and re-training of eye care personnel.
- Provision of necessary equipments and proper
maintenance of the little available ones.
41- Strict enforcement of regulation and supervision
to curtail the circulation of fake drugs. - Patient education on the need for strict
compliance with treatment regimen. - Government should subsidies the cost of glaucoma
drugs.
42CONCLUSION
- Glaucoma represents a major public
- health burden and even though the challenges of
- management in developing countries are many, they
- are not insurmountable. There is need for
concerted - and integrated efforts involving all cadres of
eye care - practitioners, patients, institutions and
governments to address this important eye disease.
43THANK YOU FOR LISTENING!
44REFERENCES
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- http//www.cehjournal.org/category/glaucoma/
- http//nurseslabs.com/3-glaucoma-nursing-care-plan
s/ - http//nursingcrib.com/nursing-notes-reviewer/medi
cal-surgical-nursing/glaucoma/ - http//eyewiki.aao.org/Glaucoma_in_the_Developing_
World - http//www.cehjournal.org/article/visual-field-tes
ting-for glaucoma-a-practical-guide/ - http//www.cehjournal.org/article/the-optic-nerve-
head-in-glaucoma/?utm_sourcehootsuite - http//www.nursingtimes.net/roles/older-people-nur
ses/glaucoma-the-silent-thief-of-sight/5075669.ful
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