Title: Generic Propecia treats Male Pattern Baldness
1Generic Propecia treats Male Pattern Baldness
2Generic Propecia Information
- Generic Propecia (generic name Finasteride) is a
drug used for the treatment of male pattern
baldness and, under the Proscar brand, enlarged
prostates. - Generic Propecia is produced by the
pharmaceutical company Merck. It is the biggest
brand for the drug named finasteride but there
are other generic brand names for this compound.
You can buy generic propecia online from online
pharmacy.
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4How does Generic Propecia work to treat male
pattern baldness?
- Generic Propecia main ingredient is a chemical
that inhibits the enzyme type II 5a-reductase.
This enzyme converts the male sex hormone
testosterone into dihydrodeoxycorticosterone
(DHT). Male pattern baldness occurs when there is
too much DHT in the scalp. Normal hair production
and maintenance processes are disrupted, and the
scalp slows down hair production or, in many
cases, stops production altogether. The culprit,
in this process, is DHT. Finasteride (Propecia)
helps increase the scalp's hair levels or reduces
hair loss by actively blocking the production of
DHT.
5Generic Propecia Effectiveness
- Generic Propecia looks to be a really effective
drug if you consider the remarkable difference
between the results experienced by the control
group compared to the ones experienced by the
test group. - According to studies, Finasteride is only
effective as long as the patient continues to
take it. If you decide to stop using Generic
Propecia, you may again experience hair loss
after about six months to a year. Unfortunately,
you will also lose the hair that you have gained
during the active treatment period, and hair loss
is again reactivated.If you do make use of
Generic Propecia, you will experience that it is
most effective on particular parts of your scalp.
You will find the best hair growth on the crown
of your head, though the medication is really
designed to restore hair and stop hair loss not
only at the crown areas, but also at the
hairline.
6How does Finasteride block DHT production?
- The male sex hormone testosterone is produced
mostly in the male's testes and adrenal glands.
Testosterone binds to a liver-produced protein
called SHBG (sex hormone-binding globulin). By
preventing testosterone from being metabolized by
the body, SHBG enables testosterone to be
transported to the body's cells. In certain parts
of the male body, primarily the prostate, skin,
and scalp, testosterone is turned into a more
potent form called 5a-dihydrotestosterone (DHT).
This conversion is made possible by 5a-reductase,
an enzyme. DHT is an amplified and more powerful
version of testosterone since it has triple to
ten times the potency of testosterone as measured
by androgen reception. DHT is made in the
following areas of the body prostate, hair
follicles, skin, and testicles. DHT is easier to
absorb that testosterone. In short, 5a-reductase
is crucial to boosting the male body's DHT
levels. DHT causes baldness because hair
follicles are very sensitive to DHT. Hair
follicles respond to elevated DHT levels by
shrinking. As a result, they produce less and
less hair or thinner hair. Given sustained and
prolonged high levels of DHT, hair follicles stop
producing hair. Similarly, high levels of DHT in
the prostate can lead to prostate enlargement. - Finasteride binds with 5a-reductase so it doesn't
convert as much testosterone into DHT.Since
Finasteride effectively 'mops up' 5a-reductase so
there are less of this enzyme in the blood
stream, the body's overall levels of DHT drops.
Lower DHT levels in the scalp and prostate
reduces or even eliminates these tissues'
reactions to elevated DHT levels. As mentioned
above, the scalp's reaction to DHT is to shrink
follicles. The prostate, on the other hand,
expands due to increased DHT. - By keeping DHT levels in check, Finasteride
enables regular follicle growth and keeps the
prostate from enlarging.
7How was finasteride discovered?
- In the mid-70s, a conference presentation by
Cornell's Julianne Imperato-McGinley showed a
link between lower levels of DHT and 5a-reductase
and smaller prostates and the absence of male
pattern baldness. Merck's head of research read
the presentation notes and went to work on
developing a compound that can lower DHT and
5a-reductase in order to treat enlarged
prostates. The resulting drug, Finasteride,
gained FDA approval for benign prostatic
hyperplasia in 1992 and male pattern baldness in
1997.