Title: Increased intracranial pressure
1Increased intracranial pressure
2Normal ICP
- 0-15 mm Hg.
- Average intracranial volume 1700 ml
- Brain (80) 1400 ml
- CSF (10) 150 ml
- Blood (10) 150 ml
3Cerebral blood flow
- CBF
-
- CPP (cerebral perfusion pressure)
- ________________________________
- CVR (cerebrovascular resistance)
4MAP 100
CPP 90 mm Hg ICP
10
Figure 26-3. Cerebral pressure. CPP MAP -
ICP. For adequate CBF, CPP should exceed 50 mm
Hg.
5Cerebral dynamics
- CPP MAP - ICP
- MAP 1/3 of (systolic MINUS diastolic blood
pressure) ADDED TO diastolic BP - Normal cerebral perfusion pressure
- 70 - 100 mm Hg.
6Cerebral dynamics
- Ischemia if CPP lt 30-40 mm Hg.
- need 60 - 70 mm Hg. MINIMALLY
- After head injury cerebral blood flow depends on
SYSTOLIC BP
7Causes of increased ICP
- 1) space occupying lesions
- 2) cerebral edema
- 3) hydrocephalus
- 4) stroke
- 5) subarachnoid hemorrhage
8Intracranial volume
9Causes of increased ICP due to changes in Brain
Parenchyma
- Head injuries
- Space occupying lesions
- - tumor
- - abscess
- Infections
- - encephalitis
- - meningitis
10Causes of increased ICP due to changes in Brain
Parenchyma
- Cerebral edema due to cardiac arrest
- Trauma related to intracranial surgery
11Causes of increased ICP due to changes in
blood/vasculature
- Hematomas
- subdural
- epidural
- intracerebral
- Subarachnoid hemorrhage
12Causes of increased ICP due to changes in
blood/vasculature
- Aneurysm
- Arteriovenous malformation (AVM)
- Thrombosis
13Causes of increased ICP due to changes in CSF
14Neurologic examination
- Mental status
- Sensation
- Cranial nerves
- Motor function
- Cerebellar function
- Reflexes
15Early signs of increased ICP
- 1) LOC restlessness, agitation, lethargy
- 2) PUPILS delayed or sluggish reaction,
unilateral changes in size - 3) MOTOR pronator drift, weakened hand grasp
16Early signs of increased ICP
- 4) HEADACHE in the early AM with nausea
vomiting - 5) SPEECH slowed or slurred
- 6) MEMORY mildly impaired
17Late signs of increased ICP
- 1) LOC difficult to arouse, decreasing Glasgow
Coma Scale - 2) PUPILS fixed or dilated
- 3) MOTOR posturing, flaccid muscles
18Posturing
19What posturing indicates..
- Decorticate posturing
- Damage to upper corticospinal tract
- Decerebrate posturing
- Brain stem damage
20Late signs of increased ICP
- 4) HEADACHE increasing with projectile vomiting
- 5) SPEECH decreasing, or with groans or moaning
- 6) VITAL SIGNS CUSHINGS TRIAD
21Cushings Triad
- reflects rising ICP with direct pressure on the
medullary center of brain - often seen in the terminal stage
- associated with irreversible brain stem damage
22Cushings Triad
- INCREASED BP WITH WIDENING PULSE PRESSURE
- BRADYCARDIA
- RESPIRATORY IRREGULARITIES
23(No Transcript)
24Changes in Vital signs with Increased ICP
25Herniation
26Herniation
27(No Transcript)
28Remember!!!!!!!
- LOC IS THE BEST AND EARLIEST INDICATOR OF
INCREASED INTRACRANIAL PRESSURE
29Treatment of increased ICP
30Treatment - Respiratory support
- hyperventilation
- increase RR TV to decrease pCO2 to 30 -35
- causes vasoconstriction of cerebral arteries
- reduces CBF increases venous return
31Treatment - Respiratory support
- adequate oxygenation
- keep pO2 gt 60
- treat ICP spikes with hyperventilation with AMBU
bag
32Nursing Measures
- suction prn NO MORE THAN 15 SECONDS!
- time limit prevents increase in CO2 which is a
potent cerebral vasodilator that can increase ICP - hyperventilate with 100 for 1 minute prior to
suctioning (if no COPD) synchronize bagging with
pt.
33Nursing Measures
- check ventilator settings
- trach care q 4 h
- monitor ABG's pulse oximetry
- oxygen as ordered
- if ICP increases during suctioning lidocaine
ETT/trach first! - ??muscle relaxant for respiratory synchrony
34Treatment - Drug therapy
- Osmotic diuretics/hyperosmolar therapy
- Mannitol 1 gr/Kg. continuous infusion
35Osmotic diuretics/hyperosmolar therapy
- administered through a filter
- keep serum osmolality 310 -315 mosm/L
36Osmotic diuretics/hyperosmolar therapy
- check serum osmolality
- glucose
- electrolytes (esp. K)
- accurate IO
- foley
37Treatment - Drug therapy
- Corticosteroids
- dexamethasone (Decadron)
- or
- methylprednisolone (Solu-Medrol)
38Treatment - Drug therapy
- must be tapered otherwise adrenal insufficiency
could develop - SE gastric irritation, stress ulcer, GI bleed
- To prevent
- H2 blockers
- antacids
39Treatment - Drug therapy
- Seizure prophylaxis
- Phenytoin (Dilantin)
- Carbamezepine (Tegretol)
40Treatment - BP control
- Goal keep systolic BP gt 90 mm Hg
- CPP gt 70 mg. Hg.
- Treatment
- hypertension when SBP gt 150 -160 or
- CPP gt 85 -100 mm Hg
- in adult who was previously normotensive
-
41Hypertension control
- IV Beta -blockers
- OR
- labetalol (Normodyne Trandate) alpha beta
blocker - Diuretics
-
42Other drugs
- muscle relaxants, muscle paralysis, sedation
- pancuronium bromide (Pavulon)
- benzodiazepines
- barbiturates
- MSO4 in small doses 1mg/hr
43Hypotension control control
- Phenylephrine hydrochloride (Neo-Synephrine)
- vasopressor
- alpha adrenergic agonist
- powerful peripheral vasoconstrictor with little
effect on heart
44Treatment - Temperature control
- control hyperthermia
- antipyretic drugs (Tylenol)
- hypothermia blanket
45Temperature
- assess Q 2 hours initiate treatment for
increase - guard against shivering Tx chlorpromazine
(Thorazine) - if on cooling blanket remove when body temp 1-2 F
above normal (99.8)
46Treatment - Seizure control
- Phenytoin (Dilantin) 100 mg TID or QID
PO/IV/tube - Carbamezepine (Tegretol)
- Phenobarbital
47Treatment - Fluid restriction
- range 900 -2500 ml/24 hours
- accurate IO IV, PO, enteral
- notice over bed
- divide into three 8 hour shifts
48Nursing Measures
- monitor Specific Gravity
- (DI S.G. 1.001-1.005 output gt 200 cc or more
for 2 consecutive hours) - administer stool softeners avoid enemas or
straining
49Treatment - CSF drainage
- ventricular drainage (into lateral ventricle for
drainage of CSF) TEMPORARY!!! - inserted through Burr hole in skull
- ventriculostomy is commonly used for
hydrocephalus R/T SAH
50Nursing measures
- closed system meticulous care to decrease
infection risk - collection bag usually level with auditory meatus
- note rate amount color of drainage
51Ventricular drainage
- HOB elevation determined by MD know correct
level! - strict asepsis
- dressing change q 48 h (prn)
- closed system
- monitor CS wbc count
52ICP Monitoring
53ICP monitoring
54ICP monitoring
55ICP monitoring
56Continuous ICP monitoring
- be familiar with equipment
- read interpret waveforms
- know what to do for atypical readings
- maintain strict asepsis
- monitor CS wbc count
57ICP monitoring
58(No Transcript)
59Treatment - Surgery
- remove or debulk lesion
- hematoma
- abscess
- tumor
- remove or debulk infarcted and necrotic tissue
60Treatment - Barbiturate coma
- when ICP does NOT respond to conventional therapy
- underlying therapeutic principle
- decreased cerebral metabolism decreased CBF
decreased ICP
61Barbiturate coma
- pentobarbital (Nembutal)
- sometimes
- thiopental sodium (Pentothal)
62Barbiturate coma
- loading dose IV
- 3-10 mg/Kg over 15 -30 minutes
- then
- 1 -2 mg/Kg/hr
- keep serum level 3 -4 mg/dL
63Barbiturate coma
- once coma induced - lose usual parameters of
neuro assessment (e.g., pupils, gag, swallowing) - need complete monitoring
- ICP Swan A line EKG, central peripheral
IV, ventilator, ETT/trach, - foley
64Barbiturate coma
- should have almost immediate effect of decreased
ICP - after pt. has had ICP lt 20 mm Hg (or whatever
protocol level) for 24 -72 hours drug tapered
65Barbiturate coma
- pentobarbital stored in body fat
- brain death criteria cannot be established until
drug cleared from body impact on organ
donation
66Nursing Managementpositioning turning
- HOB 30-45 in neutral position facilitates
venous return from head - AVOID prone
- Trendelenberg
- extreme neck flexion
- hip flexion gt 90
67Positioning turning
- use neck rolls or collar
- log roll turn Q2h skin care
- if awake, tell pt. to exhale during turning, or
moving (no Valsalva) - assist pt. in moving up in bed
- patient not to push with heels or arms or against
footboard - passive ROM
68Nursing Management
- Major objective
- identify protect patient from sudden increase
in ICP decreased CPP
69Neurological assessment
- assess baseline reassess
- compare to previous findings
- LOC
- pupil size reaction to light
- eye movement
- motor/ sensory function
70Neurologic examination
- Mental status LOC
- appearance affect, grooming, emotional status,
posture - cognition LOC, memory, attentions span,
judgement - emotional stability moods, feeling, thought
process - speech language voice quality, articulation,
content, comprehension
71Cranial nerves ocular movements
72Glasgow Coma Scale
73(No Transcript)
74(No Transcript)
75Vital signs
- assess compare with previous
- REMEMBER - VS compare poorly with early
neurological deterioration
76General nursing interventions
- maintain seizure precautions
- apply elastic stockings consecutive sequential
air boots monitor for DVT - administer basic hygiene preventive
interventions to control consequences of
immobility - No noxious stimuli! CALM environment
77General nursing interventions
- no noxious stimuli! CALM environment
- patient can hear!
- use therapeutic touch
- know medications!
- action, dosage, preparation, route, SE,
contraindications interactions
78Head injury
79Incidence
- 1 every 16 seconds
- mortality almost 50
- highest group 15 - 24 years
- MVAs
- males gt females 31
80Mechanisms of injury
81Types of brain injuries
82Main problems
- CEREBRAL EDEMA
- INCREASED ICP
- from cerebral edema expanding lesions
(hematoma)
83Mechanisms of injury
- Acceleration
- when immobile head is struck by a moving object
- Deceleration
- head is moving hits an immobile object
84Mechanisms of injury
- Acceleration - deceleration
- moving object hits the immobile head then the
head hits an immobile object - Deformation
- results in disruption of integrity of skull
85Other categorizations of head injury
- Blunt
- Penetrating
- Coup/contrecoup
86Risk factors
- Alcohol
- No seat belt
- No helmet
87Types of head injuries
88Scalp injuries
- abrasion
- scraping away
- contusion
- bruise
- laceration
- wound or tear, may bleed profusely
89Scalp injuries
- TREATMENT
- skull films
- abrasion none
- ice
- suturing
90Skull fractures
- Types
- Linear
- Comminuted
- Depressed
- Basal
91Linear fractures
- simple fracture
- crack in skull
- 70-80 of skull fractures
- treatment
- bedrest
- neuro check
92Comminuted fracture
- fragmentation of bone into many pieces or
multiple fracture lines
93Depressed skull fracture
- inward depression of the bone fragments to at
least the thickness of the skull - hair, dust, debris may be found
- dura may or may not be torn
94Depressed skull fracture
- Treatment surgery debride
- craniectomy (depressed or comminuted)
- cranioplasty
- insertion of bone or artificial graft
- may be done immediately or postponed for 3-6
months (if cerebral edema present) - DEXAMETHASONE (decadron)
95Basal skull fracture Base of skull
- may be linear, comminuted, or depressed
- can be more serious
- CSF can leak through nose/ear
rhinorrhea/otorrhea
96Basal skull fracture
- increased risk of meningitis
- appearance of blood encircled by yellowish stain
on dressing or bed linen - "halo 'sign blood encircled by CSF
97Basal Skull Fracture Anterior fossa fracture
- fracture of the Paranasal sinus
- rhinorrhea
- subconjunctival hemorrhage
- periorbital ecchymosis (raccoon's eyes)
98Basal skull fracture Anterior Fossa Fracture
99Basal skull fracture Middle fossa fracture
- associated with fracture of temporal petrous
bone involves the middle ear - otorrhea
- hemotympanum
- conductive hearing loss
100Middle fossa fracture
- may have signs of vestibular dysfunction
vertigo, nausea, nystagmus - facial nerve palsy (bell's palsy) -- appears 5-7
days after injury - ecchymosis over mastoid bone "Battle's" sign --
does not develop for 24-36 hours
101Basal Skull FractureMiddle Fossa fracture
102With skull fractures if dura torn
- ? prophylactic antibiotics
- most leaks resolve spontaneously within 7-10 days
- to aid resolution of leak LPs BID to remove 30cc
CSF
103If Dura torn
- lumbar catheter for continual drainage
- craniotomy to repair tear surgically
104Brain injuries
105Focal brain injuries
- cerebral contusion
- bruising of the surface of the brain
hemorrhagic area present -
- cerebral laceration
- actual tearing of cortical surface of brain
- (may be found with contusion)
106Focal brain injuries
- Can cause cerebral edema with increased ICP
- SS R/T anatomic area involved
- CT scan to identify contusions
- Treatment increased ICP
107Focal brain injuries
- possible rehabilitation
- management of postinjury problems seizures
108Types of diffuse brain injuries
- concussion
- diffuse axonal injury
109Concussion
- means to shake violently
- S S
- immediate unconsciousness (seconds, minutes,
hours) - momentary loss of reflexes
- momentary (few seconds) respiratory arrest
110Concussion
- possible amnesia
- headache, drowsiness, confusion, dizziness,
irritability, giddiness, visual disturbances
(seeing stars), gait disturbances
111Diffuse axonal injury
- widespread damage to axons in the white matter in
the hemispheres - R/T
- high speed acceleration - deceleration associated
with MVA's
112Diffuse axonal injury
- SS
- immediate coma
- decerebration an initially low ICP
- 94 die or remain in chronic vegetative state
long term care
113(No Transcript)
114Hematomas
115Epidural hematoma
- also called extradural hematoma
- bleeding into the potential space between the
skull dura mater - 2 of all types of head injury
- 85 also have a skull fracture
116Epidural hematoma
117Epidural hematoma
- DX CT scan
- seen most often in children young people
because the dura is less firmly attached to bone
118Epidural hematoma
- SS
- momentary unconsciousness followed by a lucid
period (few hours to 1-2 days) longer if venous
bleed involved - then
- decreased LOC
- other SS HA, seizures
119Epidural hematoma
- Treatment surgery
- Burr holes to evacuate clot ligate bleeding
vessels
120Burr holes
121Jackson - Pratt drain in Burr hole
122Subdural hematoma
- bleeding between dura mater arachnoid layer of
the meninges - causes direct pressure on the brain
- 10 -15 of head injuries develop subdural
- hematomas
123Subdural hematoma
124Subdural hematoma
125Subdural hematoma
- Diagnosis CT scan
- S S
- 3 categories based on interval between injury
appearance of ss
126Categories of subdural hematoma
- A) ACUTE within 48 hrs
- B) SUBACUTE 2 days - 2 weeks
- C) CHRONIC 2 weeks to several months
127Subdural hematoma
- associated with cerebral contusion laceration
- headache, drowsiness, slow cerebration, confusion
-- all worsen - ipsilateral pupil dilates fixed
- hemiparesis late sign
128Subdural hematoma
- elderly pts. chronic alcoholics prone to
subdurals -- r/t cerebral atrophy - treatment
- small ones medical tx
- large surgery burr holes
129Intracerebral hematoma
- bleed into cortical substance
- 2 -3 of head injuries
- R/T contusions -- tend to occur in frontal
temporal lobes
130Intracerebral hematoma
- Diagnosis CT scan
- SS
- unconsciousness, decreased LOC, HA, hemiplegia on
contralateral side, dilated pupil on side of clot - Treatment mortality high
- injury to blood vessels can cause vasospasm
131General head injury treatment
- treat all head injured patients for possible
cervical fracture - immediately immobilize neck
- patent airway but do not hyperextend
- jaw thrust maneuver
132General head injury treatment
- AIRWAY
- all unconscious head injured patients ETT to
prevent aspiration - NO CERVICAL HYPEREXTENSION
- clear nose mouth of blood, mucus, drainage
133General head injury treatment
- aspiration prior to admission possible even with
negative CXR - limit suction lt 15 seconds
- do not use nasal passage for suction until basal
skull fracture dural tear ruled out - oxygen/ventilator
134General head injury treatment
- vital signs
- assess pulses capillary refill
- peripheral IV
135General head injury treatment
- EKG monitoring
- if BP low --? occult bleeding in abdomen
- if BP high --R/T head injury -- ICP protocol
136General head injury treatment
- Labs
- CBC, electrolytes, TC, ABG's, drug screen
- Xray
- cervical spine, chest, long bones, pelvis
137General head injury treatment
- CT
- immediate if patient unconscious focal signs
are present - head to toe exam for other injuries
- Glasgow Coma scoring
138Detailed neuro exam
- LOC
- pupillary signs responses
- eye movement
- oculovestibular oculocephalic corneal
reflexes gag brain stem fx - motor responses
139Head injury
- peak swelling 72 hours after injury
140Other measures
- NG (if no basal skull fx)
- keep gastric pH _at_ 4-5
141Nutrition support
- jejunal feedings by day 7
- patient in hypercatabolic state
- as early as 24 - 48 hrs. after injury
- 140 of caloric requirements if not paralyzed
- 100 if paralyzed
- 15 protein
142(No Transcript)
143Interdisciplinary rehabilitation
- rehabilitation team approach maintenance,
prevention, restoration - Rancho Los Amigos Scale
- PT for paresis/paralysis
144Interdisciplinary rehabilitation
- OT for ADL performance evaluation deficits
- Speech therapy for communication feeding
swallowing - Neuro-opthamologist for visual deficits
- Neuropsychologist for cognitive deficits
145Interdisciplinary rehabilitation
- Urologist for bowel/bladder problems
- Psychiatrist for behavioral problems
- National Head Injury Foundation
146Intracranial hemorrhage
- Bleeding into the brain tissue or subarachnoid
space - usually due to
- head injury
- aneurysms
147Aneurysm
- Localized arterial wall dilation that develops
secondary to a weakness of the arterial wall - 90 congenital
- 80 occur in Circle of Willis
148Cerebral arteries
149Circle of Willis
150Circle of Willis
151Incidence of SAH
- 18, 000 in U.S. annually
- 20 - 40 die at initial bleed
- 1/3 of survivors have residual changes
- females gt males
- peaks in 50s
152Risk factors
- Hypertension
- Cocaine use
- Head trauma
- Congenital
153SAH
- Specific signs symptoms depends on
- location of hemorrhage
- degree of increased ICP
154Pathophysiology
- bleeding commonly stopped by formation of
fibrin-platelet plug at point of rupture by
tissue compression - within three weeks hemorrhage undergoes
re-absorption
155Pathophysiology
- serious risk of recurrent rupture 7 - 10 days
after original hemorrhage - massive hemorrhage (30 - 50 ml)
- produces rapid filling of ventricular system OR
- produces a hematoma that distorts subarachnoid
space brain tissue
156Classification
- Saccular (Berry)
- Fusiform
157Types of aneurysms
158Complications
- Rebleeding
- Vasospasm
- Hydrocephalus
159Complication - Rebleeding
- greatest cause of mortality
- peak 24 hours and in 7 - 10 days
- treatment clipping
- if no surgery Antifibrinolytic agents
(Aminocaproic Acid Amicar) to prevent clot
dissolution - SE vasospasm
160Complication - Vasospasm
- narrowing of vessel lumen
- usually in vessel adjacent to ruptured aneurysm
- may spread throughout all major vessels _at_ base of
brain - produces symptoms of ischemia
- 30 - 50 after SAH
- 65 after surgery
161Vasospasm treatment
- DRUG
- Nimodipine (nimotop)
- cerebroselective Calcium channel blocker
- 60 mg. Q 4 hour no later than 48 hours after
hemorrhage - continue for 21 days
- monitor BP carefully
162Vasospasm treatment
- DRUG
- Nicardipine (Cardene)
- alternative to Nimodipine
163Vasospasm treatment
- Intravascular volume expansion
-
- Induced arterial hypertension
- Hypervolemic - hypertensive therapy
164Vasospasm treatment
- Hypervolemic - hypertensive therapy
- increases volume pressure
- forces blood through spastic vessels
- increases flow to ischemic areas
165Vasospasm treatment
- Hypervolemic - hypertensive therapy
- keep low Hct (40) low viscosity
- albumin
- IV fluids
- keep CVP 10 mm Hg. (PCWP 18 - 20 Hg.)
166Vasospasm treatment
- Hypervolemic - hypertensive therapy
- keep SBP 150 or higher
- clipped 200
- unclipped 160
- drugs
- Dopamine, Dobutamine, Levarterenol, Metaramine
167Complication - Hydrocephalus
- caused by blood in subarachnoid space
- prevents adequate CSF circulation
- contributes to increased ICP
- teatment shunt
- ventriculoperitoneal
- ventriculoatrial
168Aneurysm clinical manifestations
- most are asymptomatic until the time of bleeding
- some are uncovered _at_ autopsy and NEVER bleed
- warning signs in 49
169Clinical manifestations
- Headache
- This is the worst headache of my life
- Localized S S depend on size location of
aneurysm
170Clinical manifestations
- Dysfunction of
- CN II optic vision
- CN III occulomotor eye movements, pupils
size, accomodation - CN V trigeminal eye movement, sensations of
head face
171Cranial nerves ocular movements
172Clinical manifestations
- Hemiparesis/hemiplegia
- Vomiting
- Seizures
- Meningeal irritation
- stiff neck
- leg back pain
173(No Transcript)
174Diagnostic studies
- Lumbar puncture
- done with caution due to increased opening
pressures (nl 50 - 180 mm H2O) - bloody CSF with Xanthochromia (hemolyzed RBCs)
- CT scan or MRI
- blood in subarachoid space, clots
- displaced structures
175Diagnostic studies
- Cerebral arteriogram
- identifies aneurysm structure location
- identifies vessels supplying aneurysm
- identifies local or general vasospasm
- outlines cerebral vascualture
- small lt 15 mm
- large 15 -25 mm
- giant 25 - 50 mm
- super giant gt 50 mm
176Cerebral arteriogram
177Treatment
- Surgery
- Corticosteroids
- Anticonvulsants
- Phenytoin (Dilantin)
- Phenobarbital
- Antihypertensives
178Treatment
- Antifibrinolytics/hemostatic agents
- Aminocaproic Acid (amicar) 24 -36 g IV Q day for
3 weeks - Analgesics/antipyretics
- Tylenol
- Tylenol with codeine
- Pituitary hormone
- Vasopressin (pitressin)
179Treatment
- Stool softener
- Electromechanical
- ventilatory support
- hypothermia blanket
- EKG monitoring
- arterial BP monitoring
180Supportive treatment
- Elevate HOB
- Subarachnoid precautions
- dim lights
- private room
- decease noise
- limit visitors
- NO Valsalva
181Supportive treatment
- Seizure precautions
- Foley
- No restraints
182Interventional radiology
- Balloon occlusion of aneurysm
- Balloon occlusion of parent vessel
- Percutaneous transfemoral approach
183Surgery
- Craniotomy
- Microsurgery
- Controlled systemic hypotension during the 5 - 10
minutes of the dissection of the aneurysm - bloodless field
- collapsed aneurysm
184Surgery
- Berry (saccular ) clip
- Fusiform wrap with special gauze acrylic wrap
185Aneurysm surgery
186Aneurysm clipping
187Arteriovenous malformation
- Congenital
- Tangles of thin walled blood vessels without
intervening capillaries - Some large others microscopic
188AVM treatment
- Neuroradiologic procedures
- embolization
- laser
- Surgery
189Neurosurgery
190Cranioplasty
- replacement of part of cranium with a plate
- metal (tantalum)
- nonmetallic material (methyl methacrylate)
- closure can be delayed for 6 months to 1 year
191Surgical approaches
- SUPRATENTORIAL
- INFRATENTORIAL
192Supratentorial
- above double fold of dura called tentorium
- incision within hairline over involved area
193Surgical approaches
- Supratentorial
- cerebrum (cerebral hemispheres)
- approach used to get at
- frontal lobe
- parietal lobe
- temporal lobe
- occipital lobe
194Supratentorial
195Infratentorial
- below tentorium
- suboccipital incision made with patient in
sitting position
196Surgical approaches
- Infratentorial
- brain stem
- midbrain
- pons
- medulla
- cerebellum
197Infratentorial
198Cranial surgery
199Stereotaxis
- precisely localizing areas in brain
- stereotactic probe or electrode passed to target
area - placement confirmed by CT scan
200Stereotaxis
- done under local anesthesia
- used to
- remove or biopsy deep, small subcortial tumors
that previously were inaccessible by routine
surgery
201Uses for Stereotaxis
- ablate lesions in extrapyramidal disorders
causing rigidity uncontrolled movements - aspirate cysts, abscesses, hematomas
- implant radioactive seeds
- interrupt pain fibers/centers
202Laser
- narrow laser beam
- excellent for removing highly vascular lesions
due to ability to simultaneously dissect,
coagulate, vaporize abnormal tissue - no bleeding into the field
203Laser
- no trauma to surrounding tissue
- allows removal of tumors proximal to delicate
cerebral
204Cryosurgery
- liquid nitrogen to produce temperatures as low as
-20 c - destroy abnormal tissue by using cold
temperatures
205Stereotactic radiosurgery
- can be performed with gamma knife, which is
actually not knife but a helmet containing
radioactive cobalt - focus so precise on malignant tissue one
treatment enough
206Stereotactic radiosurgery
- surrounding tissue not harmed
- only a few facilities have this because of its
expense - may take from 1 to 3 years lag time before lesion
is totally destroyed
207Measures to preserve cerebral function during
surgery
- HYPOTENSION
- to control cerebral blood flow during repair of
aneurysm or AVM - accomplished by
- use of sitting position
- vasodilators (sodium nitroprusside nipride)
- effects of anesthetics (halothane)
208Measures to preserve cerebral function during
surgery
- HYPOTHERMIA
- reduces oxygen consumption of brain thus
decreasing chance of neuronal damage - metabolic by-products also reduced
- accomplished by hypothermia blanket
209Measures to preserve cerebral function during
surgery
- HYPERVENTILATION
- to decrease ICP by decreasing CO2
- slows cerebral blood flow
- constricts cerebral vessels so increases venous
return - reduces intracranial volume
- accomplished by ETT ventilator
210Complications during surgery
- 1)elevated ICP controlled by
- hyperventilation
- osmotic diuretics
- dexamethasone (Decadron)
- 2) seizure activity controlled by
- phenytoin (Dilantin) (pre post op)
211Complications during surgery
- 3) infection controlled by
- aseptic techniques
- antibiotics
- 4) venous air embolism
- potential problem when surgery in sitting
position - having head higher than heart causes negative
pressure in cerebral veins venous sinuses
212Complications during surgery
- 4) venous air embolism
- air in venous system goes to right heart
- patient monitored with doppler sensor to detect
air - if air detected, surgeon identifies occludes
entry site - anesthesiologist aspirates air through the
central venous catheter
213Complications during surgery
- 4) venous air embolism
- patient vital signs are stabilized surgery
continues - if entry site of air cannot be identified,
surgery terminated patient placed in supine
position immediately monitored for transient
neurologic deficits - air embolus can be fatal
214Surgical approaches
- By Fossa
- Anterior fossa
- frontal lobe
- Middle fossa
- temporal lobe
- parietal lobe
- occipital lobe
- Posterior fossa
- brain stem cerebellum
215Surgical approaches
- Supratentorial
- Infratentorial
216Preoperative care
- antiseptic shampoos to head
- no coughing, no enemas, no leg exercises due to
increased ICP - teach relaxation techniques
- discuss tubes, monitors, appliances
(ventilator)
217Preoperative care
- hair not usually removed unless absolutely
necessary - show how to cover with stockinet
caps, scarves, hats, or a hairpiece - preop med based on specific pathology LOC
- NPO
218Preoperative care
- preop corticosteroids (dexamethasone) to control
cerebral edema - anesthesia light since the brain itself has no
pain receptors - osmotic diuretic (mannitol) may be given to aid
in decreasing increased ICP
219Preoperative care
- antibiotics prescribed if organism isolated or as
prophylaxis, esp. if ventriculostomy is
anticipated - drains (Jackson - Pratt for 24 - 48 hours)
- could be entry site for infection
220Preoperative care
- anticonvulsant
- careful neuro cognitive assessment performed
and documented to use as reference during surgery
immediately postop
221Postoperative care
- depends on specific problem
- must have following data
- neurologic status specific deficits
preoperatively - other medial problems existing preoperatively
- purpose of surgery
222Postoperative care
- Needed data
- actual procedure used
- location of the area of involvement whether a
bone flap was replaced if a large area was
evacuated - intraoperative problems
223Postoperative nursing goals
- 1) prevention recognition of complications
- 2) evaluation of patient's neurologic status
- 3) prevention, recognition, control of
increased ICP - 4) supportive care
- 5) rehabilitation
224Postoperative nursing care
- usually ICU
- hemodynamic monitoring
- many IV lines but keep fluid amount low to
prevent cerebral edema - bed have alternating pressure mattress
- hypothermia blanket to rewarm patient
225Postoperative nursing care
- IV meds immediately available for tx of
- vasospasm
- increased ICP
- HTN
- hypotension
- infection
- seizures
- cardiac arrhythmias
226Postoperative nursing care
- seizure precautions
- antiembolic stockings a sequential compression
device - ETT/ventilator/suction/oxygen
227Postoperative nursing assessment
- document LOC (glasgow coma scale)
- pupillary signs
- ocular movement
- sensory function
- motor function
- vital signs
- compare baseline data to preoperative
intraoperative data
228Postoperative nursing assessment
- neuro assessments q 15-30 minutes until stable
(or more frequently if unstable) no time frame
can be placed on this process, can vary from 4
hours to days - once patient is stable, assessments are q 1 hour
initially then every 2-4 hours
229Postoperative nursing assessment
- urine specimens to measure osmolality specific
gravity - blood for electrolytes, therapeutic drug levels,
ABGs - cultures from sputum, urine, blood, wound
sources if fever
230Proper positioning
- Supratentorial approach
- HOB elevated to 30
- turn patient to either side unless a large area
of tissue removed - if removed, patient should not lie on operated
side
231Proper positioning
- Infratentorial approach
- relatively flat with a very small pillow to neck
- flat position prevents pressure on brain stem
- can turn to either side but may not be allowed by
some surgeons to lie on back
232Proper positioning
- Infratentorial approach
- can experience dizziness so remind patient no
sudden moves (dizziness due to edema of CN VIII) - maintain position for 1 week with very gradual
elevation to 30 if tolerated
233Postoperative nursing assessment
- oral fluids
- (after 24 hours) post nausea
- if patient able to swallow (CN IX X gag
swallow) - watch for CSF leak
234Postoperative nursing assessment
- inspect eyes q 2 hours for drying or abrasions
blink corneal may be absent - periorbital swelling (48 - 72 hours eye may be
swollen shut)